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Steven J Coultrap

Showing results (31-40 of 45) with videos related to

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Nature Communications|June 8, 2017
The CaMKII holoenzyme structure in activation-competent conformationsJanette B Myers, Vincent Zaegel, Steven J Coultrap, et al.
Neuroreport|September 17, 2004
Rosiglitazone improves contextual fear conditioning in aged ratsCarmelina Gemma, Heather Stellwagen, Matthew Fister, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|May 21, 2014
Autonomous CaMKII requires further stimulation by Ca2+/calmodulin for enhancing synaptic strengthKelsey Barcomb, Isabelle Buard, Steven J Coultrap, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|June 18, 2010
CaMKII "autonomy" is required for initiating but not for maintaining neuronal long-term information storageIsabelle Buard, Steven J Coultrap, Ronald K Freund, et al.
Molecular Biology of the Cell|March 26, 2025
Aβ impairs the LTP-related movement of endogenous CaMKII but not of exogenous GFP-CaMKIICarolyn Nicole Brown, Fan-Yi Chao, Daphne Quang, et al.
Iscience|October 20, 2021
Characterization of six CaMKIIα variants found in patients with schizophreniaCarolyn Nicole Brown, Sarah G Cook, Hillary F Allen, et al.
Cell Reports|February 4, 2014
Autonomous CaMKII mediates both LTP and LTD using a mechanism for differential substrate site selectionSteven J Coultrap, Ronald K Freund, Heather O'Leary, et al.
Journal of Neurophysiology|August 31, 2007
NMDA receptor trafficking at recurrent synapses stabilizes the state of the CA3 networkJennifer L Hellier, David R Grosshans, Steven J Coultrap, et al.
The Journal of Biological Chemistry|December 3, 2017
CaMKII regulates the depalmitoylation and synaptic removal of the scaffold protein AKAP79/150 to mediate structural long-term depressionKevin M Woolfrey, Heather O'Leary, Dayton J Goodell, et al.
Iscience|October 27, 2021
GluN2B S1303 phosphorylation by CaMKII or DAPK1: no indication for involvement in ischemia or LTPJonathan E Tullis, Olivia R Buonarati, Steven J Coultrap, et al.
Pageof 5

Showing results (31-40 of 45) with videos related to

Sort By:
Pageof 5
Nature Communications|June 8, 2017
The CaMKII holoenzyme structure in activation-competent conformationsJanette B Myers, Vincent Zaegel, Steven J Coultrap, et al.
Neuroreport|September 17, 2004
Rosiglitazone improves contextual fear conditioning in aged ratsCarmelina Gemma, Heather Stellwagen, Matthew Fister, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|May 21, 2014
Autonomous CaMKII requires further stimulation by Ca2+/calmodulin for enhancing synaptic strengthKelsey Barcomb, Isabelle Buard, Steven J Coultrap, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|June 18, 2010
CaMKII "autonomy" is required for initiating but not for maintaining neuronal long-term information storageIsabelle Buard, Steven J Coultrap, Ronald K Freund, et al.
Molecular Biology of the Cell|March 26, 2025
Aβ impairs the LTP-related movement of endogenous CaMKII but not of exogenous GFP-CaMKIICarolyn Nicole Brown, Fan-Yi Chao, Daphne Quang, et al.
Iscience|October 20, 2021
Characterization of six CaMKIIα variants found in patients with schizophreniaCarolyn Nicole Brown, Sarah G Cook, Hillary F Allen, et al.
Cell Reports|February 4, 2014
Autonomous CaMKII mediates both LTP and LTD using a mechanism for differential substrate site selectionSteven J Coultrap, Ronald K Freund, Heather O'Leary, et al.
Journal of Neurophysiology|August 31, 2007
NMDA receptor trafficking at recurrent synapses stabilizes the state of the CA3 networkJennifer L Hellier, David R Grosshans, Steven J Coultrap, et al.
The Journal of Biological Chemistry|December 3, 2017
CaMKII regulates the depalmitoylation and synaptic removal of the scaffold protein AKAP79/150 to mediate structural long-term depressionKevin M Woolfrey, Heather O'Leary, Dayton J Goodell, et al.
Iscience|October 27, 2021
GluN2B S1303 phosphorylation by CaMKII or DAPK1: no indication for involvement in ischemia or LTPJonathan E Tullis, Olivia R Buonarati, Steven J Coultrap, et al.
Pageof 5