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Circulation Research
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February 4, 2012
A caveolae-targeted L-type Ca²+ channel antagonist inhibits hypertrophic signaling without reducing cardiac contractility
Catherine A Makarewich, Robert N Correll, Hui Gao, et al.
Journal of the American Heart Association
|
October 30, 2018
Outcomes Associated With a Strategy of Adjuvant Metolazone or High-Dose Loop Diuretics in Acute Decompensated Heart Failure: A Propensity Analysis
Meredith A Brisco-Bacik, Jozine M Ter Maaten, Steven R Houser, et al.
Circulation Research
|
May 26, 2012
Severe hyperhomocysteinemia promotes bone marrow-derived and resident inflammatory monocyte differentiation and atherosclerosis in LDLr/CBS-deficient mice
Daqing Zhang, Pu Fang, Xiaohua Jiang, et al.
Circulation Research
|
October 4, 2008
Ca2+ influx through T- and L-type Ca2+ channels have different effects on myocyte contractility and induce unique cardiac phenotypes
Naser Jaleel, Hiroyuki Nakayama, Xiongwen Chen, et al.
American Journal of Translational Research
|
May 22, 2012
Validation of transcatheter left ventricular electromechanical mapping for assessment of cardiac function and targeted transendocardial injection in a porcine ischemia-reperfusion model
Sharven Taghavi, Jason M Duran, Remus M Berretta, et al.
Journal of Molecular and Cellular Cardiology
|
January 22, 2004
Abnormal frequency-dependent responses represent the pathophysiologic signature of contractile failure in human myocardium
Eric I Rossman, Rebecca E Petre, Khuram W Chaudhary, et al.
Circulation Research
|
October 8, 2005
Ca2+ influx-induced sarcoplasmic reticulum Ca2+ overload causes mitochondrial-dependent apoptosis in ventricular myocytes
Xiongwen Chen, Xiaoying Zhang, Hajime Kubo, et al.
Clinical and Translational Science
|
June 17, 2014
Imatinib activates pathological hypertrophy by altering myocyte calcium regulation
Larry A Barr, Catherine A Makarewich, Remus M Berretta, et al.
Journal of Molecular and Cellular Cardiology
|
September 1, 2009
Pleiotropic effects of neutrophils on myocyte apoptosis and left ventricular remodeling during early volume overload
Mikhail A Kolpakov, Rachid Seqqat, Khadija Rafiq, et al.
Journal of Molecular and Cellular Cardiology
|
August 28, 2012
Ca(2+) influx through L-type Ca(2+) channels and transient receptor potential channels activates pathological hypertrophy signaling
Hui Gao, Fang Wang, Wei Wang, et al.
Page
of 21
Search research articles
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Showing results (91-100 of 209) with videos related to
Sort By:
Page
of 21
Circulation Research
|
February 4, 2012
A caveolae-targeted L-type Ca²+ channel antagonist inhibits hypertrophic signaling without reducing cardiac contractility
Catherine A Makarewich, Robert N Correll, Hui Gao, et al.
Journal of the American Heart Association
|
October 30, 2018
Outcomes Associated With a Strategy of Adjuvant Metolazone or High-Dose Loop Diuretics in Acute Decompensated Heart Failure: A Propensity Analysis
Meredith A Brisco-Bacik, Jozine M Ter Maaten, Steven R Houser, et al.
Circulation Research
|
May 26, 2012
Severe hyperhomocysteinemia promotes bone marrow-derived and resident inflammatory monocyte differentiation and atherosclerosis in LDLr/CBS-deficient mice
Daqing Zhang, Pu Fang, Xiaohua Jiang, et al.
Circulation Research
|
October 4, 2008
Ca2+ influx through T- and L-type Ca2+ channels have different effects on myocyte contractility and induce unique cardiac phenotypes
Naser Jaleel, Hiroyuki Nakayama, Xiongwen Chen, et al.
American Journal of Translational Research
|
May 22, 2012
Validation of transcatheter left ventricular electromechanical mapping for assessment of cardiac function and targeted transendocardial injection in a porcine ischemia-reperfusion model
Sharven Taghavi, Jason M Duran, Remus M Berretta, et al.
Journal of Molecular and Cellular Cardiology
|
January 22, 2004
Abnormal frequency-dependent responses represent the pathophysiologic signature of contractile failure in human myocardium
Eric I Rossman, Rebecca E Petre, Khuram W Chaudhary, et al.
Circulation Research
|
October 8, 2005
Ca2+ influx-induced sarcoplasmic reticulum Ca2+ overload causes mitochondrial-dependent apoptosis in ventricular myocytes
Xiongwen Chen, Xiaoying Zhang, Hajime Kubo, et al.
Clinical and Translational Science
|
June 17, 2014
Imatinib activates pathological hypertrophy by altering myocyte calcium regulation
Larry A Barr, Catherine A Makarewich, Remus M Berretta, et al.
Journal of Molecular and Cellular Cardiology
|
September 1, 2009
Pleiotropic effects of neutrophils on myocyte apoptosis and left ventricular remodeling during early volume overload
Mikhail A Kolpakov, Rachid Seqqat, Khadija Rafiq, et al.
Journal of Molecular and Cellular Cardiology
|
August 28, 2012
Ca(2+) influx through L-type Ca(2+) channels and transient receptor potential channels activates pathological hypertrophy signaling
Hui Gao, Fang Wang, Wei Wang, et al.
Page
of 21