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Steven S Scherer

Showing results (11-20 of 159) with videos related to

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Glia|September 23, 2008
Molecular mechanisms of inherited demyelinating neuropathiesSteven S Scherer, Lawrence Wrabetz
Journal of Neuromuscular Diseases|August 20, 2019
A MT-ATP6 Mutation Causes a Slowly Progressive MyeloneuropathyTanya Bardakjian, Steven S Scherer
Neurology|June 13, 2014
Ranvier revisited: novel nodal antigens stimulate interest in GBS pathogenesisHugh Willison, Steven S Scherer
Neurobiology of Disease|June 9, 2015
Activated immune response in an inherited leukodystrophy disease caused by the loss of oligodendrocyte gap junctionsSameh K Wasseff, Steven S Scherer
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|February 11, 2005
Altered ion channels in an animal model of Charcot-Marie-Tooth disease type IAJérôme J Devaux, Steven S Scherer
Neurotherapeutics : the Journal of the American Society for Experimental Neurotherapeutics|October 4, 2021
Axonal Charcot-Marie-Tooth Disease: from Common Pathogenic Mechanisms to Emerging Treatment OpportunitiesBrett A McCray, Steven S Scherer
The Journal of Comparative Neurology|December 3, 2011
Kv7.5 is the primary Kv7 subunit expressed in C-fibersChih H King, Steven S Scherer
Journal of the Peripheral Nervous System : JPNS|April 10, 2002
Recent progress on the molecular organization of myelinated axonsSteven S Scherer, Edgardo J Arroyo
Journal of Neuroimmunology|March 4, 2014
Activated microglia do not form functional gap junctions in vivoSameh K Wasseff, Steven S Scherer
Journal of the Peripheral Nervous System : JPNS|January 3, 2013
X-linked Charcot-Marie-Tooth diseaseSteven S Scherer, Kleopas A Kleopa
Pageof 16

Showing results (11-20 of 159) with videos related to

Sort By:
Pageof 16
Glia|September 23, 2008
Molecular mechanisms of inherited demyelinating neuropathiesSteven S Scherer, Lawrence Wrabetz
Journal of Neuromuscular Diseases|August 20, 2019
A MT-ATP6 Mutation Causes a Slowly Progressive MyeloneuropathyTanya Bardakjian, Steven S Scherer
Neurology|June 13, 2014
Ranvier revisited: novel nodal antigens stimulate interest in GBS pathogenesisHugh Willison, Steven S Scherer
Neurobiology of Disease|June 9, 2015
Activated immune response in an inherited leukodystrophy disease caused by the loss of oligodendrocyte gap junctionsSameh K Wasseff, Steven S Scherer
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|February 11, 2005
Altered ion channels in an animal model of Charcot-Marie-Tooth disease type IAJérôme J Devaux, Steven S Scherer
Neurotherapeutics : the Journal of the American Society for Experimental Neurotherapeutics|October 4, 2021
Axonal Charcot-Marie-Tooth Disease: from Common Pathogenic Mechanisms to Emerging Treatment OpportunitiesBrett A McCray, Steven S Scherer
The Journal of Comparative Neurology|December 3, 2011
Kv7.5 is the primary Kv7 subunit expressed in C-fibersChih H King, Steven S Scherer
Journal of the Peripheral Nervous System : JPNS|April 10, 2002
Recent progress on the molecular organization of myelinated axonsSteven S Scherer, Edgardo J Arroyo
Journal of Neuroimmunology|March 4, 2014
Activated microglia do not form functional gap junctions in vivoSameh K Wasseff, Steven S Scherer
Journal of the Peripheral Nervous System : JPNS|January 3, 2013
X-linked Charcot-Marie-Tooth diseaseSteven S Scherer, Kleopas A Kleopa
Pageof 16