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Steven S Scherer

Showing results (61-70 of 159) with videos related to

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Annals of the New York Academy of Sciences|November 1, 2017
A Unique Mutation in Connexin32 Associated with Severe, Early Onset CMTX in a Heterozygous FemaleGrace S Lin, Jonathan D Glass, Susan Shumas, et al.
Journal of Neuropathology and Experimental Neurology|August 20, 2010
Axonal pathology precedes demyelination in a mouse model of X-linked demyelinating/type I Charcot-Marie Tooth neuropathyNatalie Vavlitou, Irene Sargiannidou, Kyriaki Markoullis, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|August 2, 2002
Connexin29 is uniquely distributed within myelinating glial cells of the central and peripheral nervous systemsBruce M Altevogt, Kleopas A Kleopa, Friso R Postma, et al.
Glia|April 23, 2002
Schwann cell caveolin-1 expression increases during myelination and decreases after axotomyDaniel D Mikol, Steven S Scherer, Sara J Duckett, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|June 14, 2003
Kv3.1b is a novel component of CNS nodesJérôme Devaux, Gisèle Alcaraz, Judith Grinspan, et al.
Molecular and Cellular Neurosciences|June 23, 2006
The effects of a dominant connexin32 mutant in myelinating Schwann cellsLinda Jo Bone Jeng, Rita J Balice-Gordon, Albee Messing, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|April 17, 2009
Connexin32 mutations cause loss of function in Schwann cells and oligodendrocytes leading to PNS and CNS myelination defectsIrene Sargiannidou, Natalie Vavlitou, Sophia Aristodemou, et al.
Annals of Neurology|May 27, 2015
Intraneural GJB1 gene delivery improves nerve pathology in a model of X-linked Charcot-Marie-Tooth diseaseIrene Sargiannidou, Alexia Kagiava, Stavros Bashiardes, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|February 17, 2006
ErbB2 signaling in Schwann cells is mostly dispensable for maintenance of myelinated peripheral nerves and proliferation of adult Schwann cells after injurySuzana Atanasoski, Steven S Scherer, Erich Sirkowski, et al.
Journal of the Peripheral Nervous System : JPNS|October 20, 2019
A recurrent GARS mutation causes distal hereditary motor neuropathyDiana C Lee, Rebecca Meyer-Schuman, Chelsea Bacon, et al.
Pageof 16

Showing results (61-70 of 159) with videos related to

Sort By:
Pageof 16
Annals of the New York Academy of Sciences|November 1, 2017
A Unique Mutation in Connexin32 Associated with Severe, Early Onset CMTX in a Heterozygous FemaleGrace S Lin, Jonathan D Glass, Susan Shumas, et al.
Journal of Neuropathology and Experimental Neurology|August 20, 2010
Axonal pathology precedes demyelination in a mouse model of X-linked demyelinating/type I Charcot-Marie Tooth neuropathyNatalie Vavlitou, Irene Sargiannidou, Kyriaki Markoullis, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|August 2, 2002
Connexin29 is uniquely distributed within myelinating glial cells of the central and peripheral nervous systemsBruce M Altevogt, Kleopas A Kleopa, Friso R Postma, et al.
Glia|April 23, 2002
Schwann cell caveolin-1 expression increases during myelination and decreases after axotomyDaniel D Mikol, Steven S Scherer, Sara J Duckett, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|June 14, 2003
Kv3.1b is a novel component of CNS nodesJérôme Devaux, Gisèle Alcaraz, Judith Grinspan, et al.
Molecular and Cellular Neurosciences|June 23, 2006
The effects of a dominant connexin32 mutant in myelinating Schwann cellsLinda Jo Bone Jeng, Rita J Balice-Gordon, Albee Messing, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|April 17, 2009
Connexin32 mutations cause loss of function in Schwann cells and oligodendrocytes leading to PNS and CNS myelination defectsIrene Sargiannidou, Natalie Vavlitou, Sophia Aristodemou, et al.
Annals of Neurology|May 27, 2015
Intraneural GJB1 gene delivery improves nerve pathology in a model of X-linked Charcot-Marie-Tooth diseaseIrene Sargiannidou, Alexia Kagiava, Stavros Bashiardes, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|February 17, 2006
ErbB2 signaling in Schwann cells is mostly dispensable for maintenance of myelinated peripheral nerves and proliferation of adult Schwann cells after injurySuzana Atanasoski, Steven S Scherer, Erich Sirkowski, et al.
Journal of the Peripheral Nervous System : JPNS|October 20, 2019
A recurrent GARS mutation causes distal hereditary motor neuropathyDiana C Lee, Rebecca Meyer-Schuman, Chelsea Bacon, et al.
Pageof 16