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Stuart L Rulten

Showing results (11-20 of 21) with videos related to

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Nucleic Acids Research|July 2, 2015
The XRCC1 phosphate-binding pocket binds poly (ADP-ribose) and is required for XRCC1 functionClaire Breslin, Peter Hornyak, Andrew Ridley, et al.
DNA Repair|November 1, 2015
Versatility in phospho-dependent molecular recognition of the XRCC1 and XRCC4 DNA-damage scaffolds by aprataxin-family FHA domainsAmy L Cherry, Timothy J Nott, Geoffrey Kelly, et al.
The EMBO Journal|November 27, 2012
APLF promotes the assembly and activity of non-homologous end joining protein complexesGabrielle J Grundy, Stuart L Rulten, Zhihong Zeng, et al.
Neurobiology of Disease|October 12, 2004
Shortfalls in the peptidyl-prolyl cis-trans isomerase protein Pin1 in neurons are associated with frontotemporal dementiasJulian R Thorpe, Sabrina Mosaheb, Lida Hashemzadeh-Bonehi, et al.
Nature Communications|April 12, 2016
The Ku-binding motif is a conserved module for recruitment and stimulation of non-homologous end-joining proteinsGabrielle J Grundy, Stuart L Rulten, Raquel Arribas-Bosacoma, et al.
Nucleic Acids Research|September 20, 2013
PARP-1 dependent recruitment of the amyotrophic lateral sclerosis-associated protein FUS/TLS to sites of oxidative DNA damageStuart L Rulten, Amy Rotheray, Ryan L Green, et al.
Molecular Cell|January 8, 2011
PARP-3 and APLF function together to accelerate nonhomologous end-joiningStuart L Rulten, Anna E O Fisher, Isabelle Robert, et al.
Nature Communications|August 18, 2016
PARP3 is a sensor of nicked nucleosomes and monoribosylates histone H2B(Glu2)Gabrielle J Grundy, Luis M Polo, Zhihong Zeng, et al.
Nature|December 22, 2016
XRCC1 mutation is associated with PARP1 hyperactivation and cerebellar ataxiaNicolas C Hoch, Hana Hanzlikova, Stuart L Rulten, et al.
Cancer Research|August 24, 2016
The PARP Inhibitor AZD2461 Provides Insights into the Role of PARP3 Inhibition for Both Synthetic Lethality and Tolerability with Chemotherapy in Preclinical ModelsLenka Oplustil O'Connor, Stuart L Rulten, Aaron N Cranston, et al.
Pageof 3

Showing results (11-20 of 21) with videos related to

Sort By:
Pageof 3
Nucleic Acids Research|July 2, 2015
The XRCC1 phosphate-binding pocket binds poly (ADP-ribose) and is required for XRCC1 functionClaire Breslin, Peter Hornyak, Andrew Ridley, et al.
DNA Repair|November 1, 2015
Versatility in phospho-dependent molecular recognition of the XRCC1 and XRCC4 DNA-damage scaffolds by aprataxin-family FHA domainsAmy L Cherry, Timothy J Nott, Geoffrey Kelly, et al.
The EMBO Journal|November 27, 2012
APLF promotes the assembly and activity of non-homologous end joining protein complexesGabrielle J Grundy, Stuart L Rulten, Zhihong Zeng, et al.
Neurobiology of Disease|October 12, 2004
Shortfalls in the peptidyl-prolyl cis-trans isomerase protein Pin1 in neurons are associated with frontotemporal dementiasJulian R Thorpe, Sabrina Mosaheb, Lida Hashemzadeh-Bonehi, et al.
Nature Communications|April 12, 2016
The Ku-binding motif is a conserved module for recruitment and stimulation of non-homologous end-joining proteinsGabrielle J Grundy, Stuart L Rulten, Raquel Arribas-Bosacoma, et al.
Nucleic Acids Research|September 20, 2013
PARP-1 dependent recruitment of the amyotrophic lateral sclerosis-associated protein FUS/TLS to sites of oxidative DNA damageStuart L Rulten, Amy Rotheray, Ryan L Green, et al.
Molecular Cell|January 8, 2011
PARP-3 and APLF function together to accelerate nonhomologous end-joiningStuart L Rulten, Anna E O Fisher, Isabelle Robert, et al.
Nature Communications|August 18, 2016
PARP3 is a sensor of nicked nucleosomes and monoribosylates histone H2B(Glu2)Gabrielle J Grundy, Luis M Polo, Zhihong Zeng, et al.
Nature|December 22, 2016
XRCC1 mutation is associated with PARP1 hyperactivation and cerebellar ataxiaNicolas C Hoch, Hana Hanzlikova, Stuart L Rulten, et al.
Cancer Research|August 24, 2016
The PARP Inhibitor AZD2461 Provides Insights into the Role of PARP3 Inhibition for Both Synthetic Lethality and Tolerability with Chemotherapy in Preclinical ModelsLenka Oplustil O'Connor, Stuart L Rulten, Aaron N Cranston, et al.
Pageof 3