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Sucharit Bhakdi

Showing results (21-30 of 54) with videos related to

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Infection and Immunity|October 24, 2003
The streptococcal exotoxin streptolysin O activates mast cells to produce tumor necrosis factor alpha by p38 mitogen-activated protein kinase- and protein kinase C-dependent pathwaysMichael Stassen, Christian Müller, Christoph Richter, et al.
Membranes|February 25, 2022
Influence of Anoctamin-4 and -9 on ADAM10 and ADAM17 Sheddase FunctionSinje Leitzke, Jana Seidel, Björn Ahrens, et al.
Critical Care Medicine|April 10, 2002
Early albumin infusion improves global and local hemodynamics and reduces inflammatory response in hemorrhagic shockGeorg Horstick, Michael Lauterbach, Tibor Kempf, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|April 7, 2006
Why Escherichia coli alpha-hemolysin induces calcium oscillations in mammalian cells--the pore is on its ownAndreas Koschinski, Holger Repp, Baris Unver, et al.
Blood|September 14, 2011
Digestive vacuoles of Plasmodium falciparum are selectively phagocytosed by and impair killing function of polymorphonuclear leukocytesPrasad Dasari, Karina Reiss, Klaus Lingelbach, et al.
Journal of Structural Biology|October 31, 2002
Interaction of the Vibrio cholerae cytolysin (VCC) with cholesterol, some cholesterol esters, and cholesterol derivatives: a TEM studyJ Robin Harris, Sucharit Bhakdi, Ulrich Meissner, et al.
Thrombosis and Haemostasis|January 25, 2008
No effect of C-reactive protein on early atherosclerosis in LDLR-/- / human C-reactive protein transgenic miceMichael Torzewski, Kurt Reifenberg, Fei Cheng, et al.
Atherosclerosis|March 10, 2009
Investigation of Sudan IV staining areas in aortas of infants and children: possible prelesional stages of atherogenesisMichael Torzewski, Bianca Navarro, Fei Cheng, et al.
Biochemical and Biophysical Research Communications|June 6, 2009
Pore-forming toxins activate MAPK p38 by causing loss of cellular potassiumNicole Kloft, Tim Busch, Claudia Neukirch, et al.
FEBS Letters|December 23, 2008
Elimination of a bacterial pore-forming toxin by sequential endocytosis and exocytosisMatthias Husmann, Erik Beckmann, Klaus Boller, et al.
Pageof 6

Showing results (21-30 of 54) with videos related to

Sort By:
Pageof 6
Infection and Immunity|October 24, 2003
The streptococcal exotoxin streptolysin O activates mast cells to produce tumor necrosis factor alpha by p38 mitogen-activated protein kinase- and protein kinase C-dependent pathwaysMichael Stassen, Christian Müller, Christoph Richter, et al.
Membranes|February 25, 2022
Influence of Anoctamin-4 and -9 on ADAM10 and ADAM17 Sheddase FunctionSinje Leitzke, Jana Seidel, Björn Ahrens, et al.
Critical Care Medicine|April 10, 2002
Early albumin infusion improves global and local hemodynamics and reduces inflammatory response in hemorrhagic shockGeorg Horstick, Michael Lauterbach, Tibor Kempf, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|April 7, 2006
Why Escherichia coli alpha-hemolysin induces calcium oscillations in mammalian cells--the pore is on its ownAndreas Koschinski, Holger Repp, Baris Unver, et al.
Blood|September 14, 2011
Digestive vacuoles of Plasmodium falciparum are selectively phagocytosed by and impair killing function of polymorphonuclear leukocytesPrasad Dasari, Karina Reiss, Klaus Lingelbach, et al.
Journal of Structural Biology|October 31, 2002
Interaction of the Vibrio cholerae cytolysin (VCC) with cholesterol, some cholesterol esters, and cholesterol derivatives: a TEM studyJ Robin Harris, Sucharit Bhakdi, Ulrich Meissner, et al.
Thrombosis and Haemostasis|January 25, 2008
No effect of C-reactive protein on early atherosclerosis in LDLR-/- / human C-reactive protein transgenic miceMichael Torzewski, Kurt Reifenberg, Fei Cheng, et al.
Atherosclerosis|March 10, 2009
Investigation of Sudan IV staining areas in aortas of infants and children: possible prelesional stages of atherogenesisMichael Torzewski, Bianca Navarro, Fei Cheng, et al.
Biochemical and Biophysical Research Communications|June 6, 2009
Pore-forming toxins activate MAPK p38 by causing loss of cellular potassiumNicole Kloft, Tim Busch, Claudia Neukirch, et al.
FEBS Letters|December 23, 2008
Elimination of a bacterial pore-forming toxin by sequential endocytosis and exocytosisMatthias Husmann, Erik Beckmann, Klaus Boller, et al.
Pageof 6