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Science Advances|January 12, 2022
RAD51AP2 is required for efficient meiotic recombination between X and Y chromosomesHui Ma, Tao Li, Xuefeng Xie, et al.
Human Reproduction (Oxford, England)|May 8, 2022
Biallelic HFM1 variants cause non-obstructive azoospermia with meiotic arrest in humans by impairing crossover formation to varying degreesXuefeng Xie, Ghulam Murtaza, Yang Li, et al.
Cell Discovery|August 23, 2023
A novel recombination protein C12ORF40/REDIC1 is required for meiotic crossover formationSuixing Fan, Yuewen Wang, Hanwei Jiang, et al.
American Journal of Human Genetics|January 28, 2021
Homozygous mutations in C14orf39/SIX6OS1 cause non-obstructive azoospermia and premature ovarian insufficiency in humansSuixing Fan, Yuying Jiao, Ranjha Khan, et al.
Iscience|July 24, 2023
Loss-of-function variants in KCTD19 cause non-obstructive azoospermia in humansJunyan Liu, Fazal Rahim, Jianteng Zhou, et al.
Genetics in Medicine : Official Journal of the American College of Medical Genetics|August 31, 2018
Correction: A homozygous FANCM frameshift pathogenic variant causes male infertilityHao Yin, Hui Ma, Sajjad Hussain, et al.
Science Bulletin|February 3, 2023
A TOP6BL mutation abolishes meiotic DNA double-strand break formation and causes human infertilityYuying Jiao, Suixing Fan, Nazish Jabeen, et al.
Genetics in Medicine : Official Journal of the American College of Medical Genetics|June 14, 2018
A homozygous FANCM frameshift pathogenic variant causes male infertilityHao Yin, Hui Ma, Sajjad Hussain, et al.
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