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ABOUT JoVE

Overview Leadership Blog JoVE Help Center

AUTHORS

Publishing Process Editorial Board Scope & Policies Peer Review FAQ Submit

LIBRARIANS

Testimonials Subscriptions Access Resources Library Advisory Board FAQ

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JoVE Journal Methods Collections JoVE Encyclopedia of Experiments Archive

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JoVE Core JoVE Business JoVE Science Education JoVE Lab Manual Faculty Resource Center Faculty Site

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Sujith Dassanayaka

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Toxicological Sciences : an Official Journal of the Society of Toxicology|October 31, 2021

Chronic Benzene Exposure Aggravates Pressure Overload-Induced Cardiac DysfunctionIgor N Zelko, Sujith Dassanayaka, Marina V Malovichko, et al.

The Biochemical Journal|January 29, 2015

High glucose induces mitochondrial dysfunction independently of protein O-GlcNAcylationSujith Dassanayaka, Ryan D Readnower, Joshua K Salabei, et al.

The Journal of Thoracic and Cardiovascular Surgery|October 22, 2013

Insights into the mechanism(s) of von Willebrand factor degradation during mechanical circulatory supportCarlo R Bartoli, Sujith Dassanayaka, Kenneth R Brittian, et al.

Journal of Molecular and Cellular Cardiology|April 9, 2018

Deficiency of aldose reductase exacerbates early pressure overload-induced cardiac dysfunction and autophagy in miceShahid P Baba, Deqing Zhang, Mahavir Singh, et al.

Redox Biology|June 10, 2018

Cardiac-specific overexpression of aldehyde dehydrogenase 2 exacerbates cardiac remodeling in response to pressure overloadSujith Dassanayaka, Yuting Zheng, Andrew A Gibb, et al.

Basic Research in Cardiology|June 2, 2019

E2f1 deletion attenuates infarct-induced ventricular remodeling without affecting O-GlcNAcylationSujith Dassanayaka, Kenneth R Brittian, Andrea Jurkovic, et al.

Basic Research in Cardiology|March 17, 2017

Cardiomyocyte Ogt limits ventricular dysfunction in mice following pressure overload without affecting hypertrophySujith Dassanayaka, Robert E Brainard, Lewis J Watson, et al.

Plos One|November 30, 2020

Cardiomyocyte Oga haploinsufficiency increases O-GlcNAcylation but hastens ventricular dysfunction following myocardial infarctionSujith Dassanayaka, Kenneth R Brittian, Bethany W Long, et al.

American Journal of Physiology. Heart and Circulatory Physiology|September 17, 2021

Cardiac PANK1 deletion exacerbates ventricular dysfunction during pressure overloadTimothy N Audam, Caitlin M Howard, Lauren F Garrett, et al.

Journal of Molecular and Cellular Cardiology|July 12, 2025

Pyruvate kinase splice variants in fibroblasts influence cardiac remodeling after myocardial infarction in male miceCollin K Wells, Daniel C Nguyen, Robert E Brainard, et al.

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Search research articles

Showing results (11-20 of 20) with videos related to

Sort By:

Pageof 2

You have reached the last page of results.This site can display upto 20 results.

Toxicological Sciences : an Official Journal of the Society of Toxicology|October 31, 2021

Chronic Benzene Exposure Aggravates Pressure Overload-Induced Cardiac DysfunctionIgor N Zelko, Sujith Dassanayaka, Marina V Malovichko, et al.

The Biochemical Journal|January 29, 2015

High glucose induces mitochondrial dysfunction independently of protein O-GlcNAcylationSujith Dassanayaka, Ryan D Readnower, Joshua K Salabei, et al.

The Journal of Thoracic and Cardiovascular Surgery|October 22, 2013

Insights into the mechanism(s) of von Willebrand factor degradation during mechanical circulatory supportCarlo R Bartoli, Sujith Dassanayaka, Kenneth R Brittian, et al.

Journal of Molecular and Cellular Cardiology|April 9, 2018

Deficiency of aldose reductase exacerbates early pressure overload-induced cardiac dysfunction and autophagy in miceShahid P Baba, Deqing Zhang, Mahavir Singh, et al.

Redox Biology|June 10, 2018

Cardiac-specific overexpression of aldehyde dehydrogenase 2 exacerbates cardiac remodeling in response to pressure overloadSujith Dassanayaka, Yuting Zheng, Andrew A Gibb, et al.

Basic Research in Cardiology|June 2, 2019

E2f1 deletion attenuates infarct-induced ventricular remodeling without affecting O-GlcNAcylationSujith Dassanayaka, Kenneth R Brittian, Andrea Jurkovic, et al.

Basic Research in Cardiology|March 17, 2017

Cardiomyocyte Ogt limits ventricular dysfunction in mice following pressure overload without affecting hypertrophySujith Dassanayaka, Robert E Brainard, Lewis J Watson, et al.

Plos One|November 30, 2020

Cardiomyocyte Oga haploinsufficiency increases O-GlcNAcylation but hastens ventricular dysfunction following myocardial infarctionSujith Dassanayaka, Kenneth R Brittian, Bethany W Long, et al.

American Journal of Physiology. Heart and Circulatory Physiology|September 17, 2021

Cardiac PANK1 deletion exacerbates ventricular dysfunction during pressure overloadTimothy N Audam, Caitlin M Howard, Lauren F Garrett, et al.

Journal of Molecular and Cellular Cardiology|July 12, 2025

Pyruvate kinase splice variants in fibroblasts influence cardiac remodeling after myocardial infarction in male miceCollin K Wells, Daniel C Nguyen, Robert E Brainard, et al.

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