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Sulayman D Dib-Hajj

Showing results (1-10 of 184) with videos related to

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Annual Review of Neuroscience|February 1, 2019
Sodium Channels in Human Pain Disorders: Genetics and PharmacogenomicsSulayman D Dib-Hajj, Stephen G Waxman
Science Translational Medicine|August 15, 2014
Translational pain research: Lessons from genetics and genomicsSulayman D Dib-Hajj, Stephen G Waxman
The Journal of Clinical Investigation|July 1, 2024
Disordered but effective: short linear motifs as gene therapy targets for hyperexcitability disordersSulayman D Dib-Hajj, Stephen G Waxman
Neuron|September 7, 2023
Na<sub>V</sub>1.7: A central role in painStephen G Waxman, Sulayman D Dib-Hajj
Annals of Neurology|June 2, 2005
Erythromelalgia: a hereditary pain syndrome enters the molecular eraStephen G Waxman, Sulayman D Dib-Hajj
Neuroscience Letters|September 7, 2010
Isoform-specific and pan-channel partners regulate trafficking and plasma membrane stability; and alter sodium channel gating propertiesSulayman D Dib-Hajj, Stephen G Waxman
Molecular Brain|August 9, 2018
Multiple myosin motors interact with sodium/potassium-ATPase alpha 1 subunitsBhagirathi Dash, Sulayman D Dib-Hajj, Stephen G Waxman
Molecular Pain|June 10, 2010
Effects of ranolazine on wild-type and mutant hNav1.7 channels and on DRG neuron excitabilityMark Estacion, Stephen G Waxman, Sulayman D Dib-Hajj
Molecular Neurobiology|November 14, 2002
Structure of the sodium channel gene SCN11A: evidence for intron-to-exon conversion model and implications for gene evolutionSulayman D Dib-Hajj, Lynda Tyrrell, Stephen G Waxman
Molecular Pharmacology|April 19, 2002
Critical molecular determinants of voltage-gated sodium channel sensitivity to mu-conotoxins GIIIA/BTheodore R Cummins, Fabio Aglieco, Sulayman D Dib-Hajj
Pageof 19

Showing results (1-10 of 184) with videos related to

Sort By:
Pageof 19
Annual Review of Neuroscience|February 1, 2019
Sodium Channels in Human Pain Disorders: Genetics and PharmacogenomicsSulayman D Dib-Hajj, Stephen G Waxman
Science Translational Medicine|August 15, 2014
Translational pain research: Lessons from genetics and genomicsSulayman D Dib-Hajj, Stephen G Waxman
The Journal of Clinical Investigation|July 1, 2024
Disordered but effective: short linear motifs as gene therapy targets for hyperexcitability disordersSulayman D Dib-Hajj, Stephen G Waxman
Neuron|September 7, 2023
Na<sub>V</sub>1.7: A central role in painStephen G Waxman, Sulayman D Dib-Hajj
Annals of Neurology|June 2, 2005
Erythromelalgia: a hereditary pain syndrome enters the molecular eraStephen G Waxman, Sulayman D Dib-Hajj
Neuroscience Letters|September 7, 2010
Isoform-specific and pan-channel partners regulate trafficking and plasma membrane stability; and alter sodium channel gating propertiesSulayman D Dib-Hajj, Stephen G Waxman
Molecular Brain|August 9, 2018
Multiple myosin motors interact with sodium/potassium-ATPase alpha 1 subunitsBhagirathi Dash, Sulayman D Dib-Hajj, Stephen G Waxman
Molecular Pain|June 10, 2010
Effects of ranolazine on wild-type and mutant hNav1.7 channels and on DRG neuron excitabilityMark Estacion, Stephen G Waxman, Sulayman D Dib-Hajj
Molecular Neurobiology|November 14, 2002
Structure of the sodium channel gene SCN11A: evidence for intron-to-exon conversion model and implications for gene evolutionSulayman D Dib-Hajj, Lynda Tyrrell, Stephen G Waxman
Molecular Pharmacology|April 19, 2002
Critical molecular determinants of voltage-gated sodium channel sensitivity to mu-conotoxins GIIIA/BTheodore R Cummins, Fabio Aglieco, Sulayman D Dib-Hajj
Pageof 19