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Oxidative Medicine and Cellular Longevity|February 24, 2021
Calcitriol Supplementation Ameliorates Microvascular Endothelial Dysfunction in Vitamin D-Deficient Diabetic Rats by Upregulating the Vascular eNOS Protein Expression and Reducing Oxidative StressChee Lee Wee, Siti Safiah Mokhtar, Kirnpal Kaur Banga Singh, et al.
Acta Pharmacologica Sinica|December 6, 2002
Contents of four active components in different commercial crude drugs and preparations of danshen (Salvia miltiorrhiza)Hui Zhang, Chen Yu, Jing-Ying Jia, et al.
Cardiovascular Research|January 21, 2011
Role of sulfhydryl-dependent dimerization of soluble guanylyl cyclase in relaxation of porcine coronary artery to nitric oxideXiaoxu Zheng, Lei Ying, Juan Liu, et al.
Critical Reviews in Food Science and Nutrition|July 13, 2022
Exploration of natural flavones' bioactivity and bioavailability in chronic inflammation induced-type-2 diabetes mellitusLingchao Miao, Conghui Liu, Meang Sam Cheong, et al.
The Journal of Pharmacology and Experimental Therapeutics|March 25, 2010
Endothelium-derived nitric oxide inhibits the relaxation of the porcine coronary artery to natriuretic peptides by desensitizing big conductance calcium-activated potassium channels of vascular smooth muscleChao Fan Liang, Alice Lai Shan Au, Susan Wai Sum Leung, et al.
Nitric Oxide : Biology and Chemistry|January 16, 2016
Endothelium dependent hyperpolarization-type relaxation compensates for attenuated nitric oxide-mediated responses in subcutaneous arteries of diabetic patientsSiti Safiah Mokhtar, Paul M Vanhoutte, Susan Wai Sum Leung, et al.
American Journal of Physiology. Heart and Circulatory Physiology|June 8, 2014
cIMP synthesized by sGC as a mediator of hypoxic contraction of coronary arteriesZhengju Chen, Xu Zhang, Lei Ying, et al.
Acta Pharmacologica Sinica|February 24, 2021
MicroRNA-17-3p suppresses NF-κB-mediated endothelial inflammation by targeting NIK and IKKβ binding proteinYin Cai, Yu Zhang, Hui Chen, et al.
Oxidative Medicine and Cellular Longevity|June 13, 2022
Protective Effects of Amauroderma rugosum on Doxorubicin-Induced Cardiotoxicity through Suppressing Oxidative Stress, Mitochondrial Dysfunction, Apoptosis, and Activating Akt/mTOR and Nrf2/HO-1 Signaling PathwaysJingjing Li, Yanfen Cheng, Renkai Li, et al.
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