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Journal of Neurophysiology|June 30, 2006
Inhibitory postsynaptic potentials in lumbar motoneurons remain depolarizing after neonatal spinal cord transection in the ratCéline Jean-Xavier, Jean-François Pflieger, Sylvie Liabeuf, et al.
Frontiers in Molecular Neuroscience|February 20, 2026
KCC2 phosphorylation dynamics relate to oligomerization during development and after spinal cord injurySylvie Liabeuf, Cecile Brocard, Jacques-Olivier Coq, et al.
Elife|December 10, 2019
Calpain fosters the hyperexcitability of motoneurons after spinal cord injury and leads to spasticityVanessa Plantier, Irene Sanchez-Brualla, Nejada Dingu, et al.
The European Journal of Neuroscience|January 25, 2011
Contribution of the potassium-chloride co-transporter KCC2 to the modulation of lumbar spinal networks in miceAurélie Stil, Céline Jean-Xavier, Sylvie Liabeuf, et al.
Nature Medicine|March 15, 2016
Cleavage of Na(+) channels by calpain increases persistent Na(+) current and promotes spasticity after spinal cord injuryCécile Brocard, Vanessa Plantier, Pascale Boulenguez, et al.
Proceedings of the National Academy of Sciences of the United States of America|December 19, 2012
Activation of 5-HT2A receptors upregulates the function of the neuronal K-Cl cotransporter KCC2Rémi Bos, Karina Sadlaoud, Pascale Boulenguez, et al.
Neuroscience|August 28, 2017
Activation of 5-HT2A Receptors Restores KCC2 Function and Reduces Neuropathic Pain after Spinal Cord InjuryIrene Sánchez-Brualla, Pascale Boulenguez, Cécile Brocard, et al.
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