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Science (New York, N.Y.)|December 5, 1980
Epileptiform burst afterhyperolarization: calcium-dependent potassium potential in hippocampal CA1 pyramidal cellsB E Alger, R A NicollThe Journal of Physiology|August 15, 1996
Increased neuronal excitability during depolarization-induced suppression of inhibition in rat hippocampusJ J Wagner, B E AlgerHippocampus|January 1, 1996
Homosynaptic LTD and depotentiation: do they differ in name only?J J Wagner, B E AlgerJournal of Neurophysiology|February 1, 1994
GTP modulates run-up of whole-cell Ca2+ channel current in a Ca(2+)-dependent mannerJ J Wagner, B E AlgerThe Journal of Neuroscience : the Official Journal of the Society for Neuroscience|February 1, 1995
GABAergic and developmental influences on homosynaptic LTD and depotentiation in rat hippocampusJ J Wagner, B E AlgerJournal of Neurophysiology|July 1, 1986
Whole-cell voltage-clamp study of the fading of GABA-activated currents in acutely dissociated hippocampal neuronsJ R Huguenard, B E AlgerThe Journal of Physiology|July 1, 1982
Pharmacological evidence for two kinds of GABA receptor on rat hippocampal pyramidal cells studied in vitroB E Alger, R A NicollBrain Research|January 29, 1993
Perfusion with high potassium plus glutamate can cause LTP erasure or persistent loss of neuronal responsiveness in the CA1 region of the hippocampal sliceC M Harrison, B E AlgerScience (New York, N.Y.)|October 30, 1981
Brain aging correlates: retardation by hormonal-pharmacological treatmentsP W Landfield, R K Baskin, T A PitlerThe Journal of Neuroscience : the Official Journal of the Society for Neuroscience|May 1, 1984
Electrophysiological mechanisms of kainic acid-induced epileptiform activity in the rat hippocampal sliceR S Fisher, B E AlgerPageof 7