T C Saido - JoVE Visualize

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Biochemical and Biophysical Research Communications|June 26, 1995

Modified amyloid beta protein ending at 42 or 40 with different solubility accumulates in the brain of Alzheimer's diseaseY Harigaya, M Shoji, T Kawarabayashi, et al.

FEBS Letters|June 13, 1994

Distinct kinetics of subunit autolysis in mammalian m-calpain activationT C Saido, S Nagao, M Shiramine, et al.

FEBS Letters|February 13, 1995

UCN-01, an anti-tumor drug, is a selective inhibitor of the conventional PKC subfamilyK Mizuno, K Noda, Y Ueda, et al.

Journal of Neurology, Neurosurgery, and Psychiatry|June 18, 2004

Novel amyloid precursor protein gene missense mutation (D678N) in probable familial Alzheimer's diseaseY Wakutani, K Watanabe, Y Adachi, et al.

The Biochemical Journal|October 8, 1999

Membrane-anchored metalloprotease MDC9 has an alpha-secretase activity responsible for processing the amyloid precursor proteinH Koike, S Tomioka, H Sorimachi, et al.

The Journal of Biological Chemistry|May 13, 1999

Calpastatin is up-regulated in response to hypoxia and is a suicide substrate to calpain after neonatal cerebral hypoxia-ischemiaK Blomgren, U Hallin, A L Andersson, et al.

The Journal of Biological Chemistry|May 29, 2000

Activation of calpain I converts excitotoxic neuron death into a caspase-independent cell deathS Lankiewicz, C Marc Luetjens, N Truc Bui, et al.

Cancer|December 18, 2001

Calpain-dependent proteolysis of merlin occurs by oxidative stress in meningiomas: a novel hypothesis of tumorigenesisT Kaneko, T Yamashima, Y Tohma, et al.

The European Journal of Neuroscience|September 1, 1996

Transient brain ischaemia provokes Ca2+, PIP2 and calpain responses prior to delayed neuronal death in monkeysT Yamashima, T C Saido, M Takita, et al.

The American Journal of Pathology|July 6, 2000

Age-related amyloid beta deposition in transgenic mice overexpressing both Alzheimer mutant presenilin 1 and amyloid beta precursor protein Swedish mutant is not associated with global neuronal lossA Takeuchi, M C Irizarry, K Duff, et al.

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Showing results (101-110 of 129) with videos related to

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Pageof 13

Biochemical and Biophysical Research Communications|June 26, 1995

Modified amyloid beta protein ending at 42 or 40 with different solubility accumulates in the brain of Alzheimer's diseaseY Harigaya, M Shoji, T Kawarabayashi, et al.

FEBS Letters|June 13, 1994

Distinct kinetics of subunit autolysis in mammalian m-calpain activationT C Saido, S Nagao, M Shiramine, et al.

FEBS Letters|February 13, 1995

UCN-01, an anti-tumor drug, is a selective inhibitor of the conventional PKC subfamilyK Mizuno, K Noda, Y Ueda, et al.

Journal of Neurology, Neurosurgery, and Psychiatry|June 18, 2004

Novel amyloid precursor protein gene missense mutation (D678N) in probable familial Alzheimer's diseaseY Wakutani, K Watanabe, Y Adachi, et al.

The Biochemical Journal|October 8, 1999

Membrane-anchored metalloprotease MDC9 has an alpha-secretase activity responsible for processing the amyloid precursor proteinH Koike, S Tomioka, H Sorimachi, et al.

The Journal of Biological Chemistry|May 13, 1999

Calpastatin is up-regulated in response to hypoxia and is a suicide substrate to calpain after neonatal cerebral hypoxia-ischemiaK Blomgren, U Hallin, A L Andersson, et al.

The Journal of Biological Chemistry|May 29, 2000

Activation of calpain I converts excitotoxic neuron death into a caspase-independent cell deathS Lankiewicz, C Marc Luetjens, N Truc Bui, et al.

Cancer|December 18, 2001

Calpain-dependent proteolysis of merlin occurs by oxidative stress in meningiomas: a novel hypothesis of tumorigenesisT Kaneko, T Yamashima, Y Tohma, et al.

The European Journal of Neuroscience|September 1, 1996

Transient brain ischaemia provokes Ca2+, PIP2 and calpain responses prior to delayed neuronal death in monkeysT Yamashima, T C Saido, M Takita, et al.

The American Journal of Pathology|July 6, 2000

Age-related amyloid beta deposition in transgenic mice overexpressing both Alzheimer mutant presenilin 1 and amyloid beta precursor protein Swedish mutant is not associated with global neuronal lossA Takeuchi, M C Irizarry, K Duff, et al.

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