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Journal of Immunology (Baltimore, Md. : 1950)|November 15, 1986
Accessory cell-T cell interactions involved in anti-CD3-induced T4 and T8 cell proliferation: analysis with monoclonal antibodiesT D Geppert, P E LipskyJournal of Immunology (Baltimore, Md. : 1950)|January 15, 1987
Dissection of defective antigen presentation by interferon-gamma-treated fibroblastsT D Geppert, P E LipskyEuropean Journal of Immunology|June 1, 1992
Engagement of class I major histocompatibility complex molecules by cell surface CD8 delivers an activation signalT D Geppert, H Nguyen, P E LipskyJournal of Immunology (Baltimore, Md. : 1950)|January 15, 1988
Enhancement of antigen- and mitogen-induced human T lymphocyte proliferation by tumor necrosis factor-alphaS Yokota, T D Geppert, P E LipskyMolecular Immunology|February 1, 1997
Structural analysis of class I MHC molecules: the cytoplasmic domain is not required for cytoskeletal association, aggregation and internalizationH Gur, T D Geppert, P E LipskyMolecular and Cellular Biology|October 29, 1997
Jun N-terminal kinase/stress-activated protein kinase (JNK/SAPK) is required for lipopolysaccharide stimulation of tumor necrosis factor alpha (TNF-alpha) translation: glucocorticoids inhibit TNF-alpha translation by blocking JNK/SAPKJ L Swantek, M H Cobb, T D GeppertCellular Immunology|December 1, 1989
Precursor frequency of human T4 cells responding to stimulation through the CD3 molecular complex: role of various cytokines in promoting growth and IL2 productionJ B Vine, T D Geppert, P E LipskyJournal of Immunology (Baltimore, Md. : 1950)|October 15, 1988
T4 cell activation by immobilized phytohemagglutinin: differential capacity to induce IL-2 responsiveness and IL-2 productionJ B Vine, T D Geppert, P E LipskyMolecular Medicine (Cambridge, Mass.)|November 1, 1994
Lipopolysaccharide signals activation of tumor necrosis factor biosynthesis through the ras/raf-1/MEK/MAPK pathwayT D Geppert, C E Whitehurst, P Thompson, et al.Cellular Immunology|February 12, 1999
The cytoplasmic and the transmembrane domains are not sufficient for class I MHC signal transductionH Gur, T D Geppert, M C Wacholtz, et al.Pageof 4