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Showing results (61-70 of 78) with videos related to

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Neuropathology and Applied Neurobiology|March 18, 2005
Extended pathoanatomical studies point to a consistent affection of the thalamus in spinocerebellar ataxia type 2U Rüb, D Del Turco, K Bürk, et al.
Neuropathology and Applied Neurobiology|February 8, 2012
Spinocerebellar ataxia type 1 (SCA1): new pathoanatomical and clinico-pathological insightsU Rüb, K Bürk, D Timmann, et al.
Neuropathology and Applied Neurobiology|November 7, 2006
Degeneration of ingestion-related brainstem nuclei in spinocerebellar ataxia type 2, 3, 6 and 7U Rüb, E R Brunt, E Petrasch-Parwez, et al.
Neuropathology and Applied Neurobiology|January 29, 2008
Involvement of the auditory brainstem system in spinocerebellar ataxia type 2 (SCA2), type 3 (SCA3) and type 7 (SCA7)F Hoche, K Seidel, E R Brunt, et al.
Cerebellum (London, England)|December 27, 2011
Pathoanatomy of cerebellar degeneration in spinocerebellar ataxia type 2 (SCA2) and type 3 (SCA3)W Scherzed, E R Brunt, H Heinsen, et al.
Journal of Neural Transmission (Vienna, Austria : 1996)|March 24, 2005
Spinocerebellar ataxias types 2 and 3: degeneration of the pre-cerebellar nuclei isolates the three phylogenetically defined regions of the cerebellumU Rüb, K Gierga, E R Brunt, et al.
Neurology|August 9, 2006
Diffusion tensor MRI shows abnormal brainstem crossing fibers associated with ROBO3 mutationsN L Sicotte, G Salamon, D W Shattuck, et al.
Neuropathology and Applied Neurobiology|February 12, 2009
Spinocerebellar ataxia type 6 (SCA6): neurodegeneration goes beyond the known brain predilection sitesK Gierga, H J Schelhaas, E R Brunt, et al.
Neurology|October 13, 2004
Damage to the reticulotegmental nucleus of the pons in spinocerebellar ataxia type 1, 2, and 3U Rüb, K Bürk, L Schöls, et al.
Neuropathology and Applied Neurobiology|November 1, 2007
Spinocerebellar ataxia type 7 (SCA7): widespread brain damage in an adult-onset patient with progressive visual impairments in comparison with an adult-onset patient without visual impairmentsU Rüb, E R Brunt, K Seidel, et al.
Pageof 8

Showing results (61-70 of 78) with videos related to

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Pageof 8
Neuropathology and Applied Neurobiology|March 18, 2005
Extended pathoanatomical studies point to a consistent affection of the thalamus in spinocerebellar ataxia type 2U Rüb, D Del Turco, K Bürk, et al.
Neuropathology and Applied Neurobiology|February 8, 2012
Spinocerebellar ataxia type 1 (SCA1): new pathoanatomical and clinico-pathological insightsU Rüb, K Bürk, D Timmann, et al.
Neuropathology and Applied Neurobiology|November 7, 2006
Degeneration of ingestion-related brainstem nuclei in spinocerebellar ataxia type 2, 3, 6 and 7U Rüb, E R Brunt, E Petrasch-Parwez, et al.
Neuropathology and Applied Neurobiology|January 29, 2008
Involvement of the auditory brainstem system in spinocerebellar ataxia type 2 (SCA2), type 3 (SCA3) and type 7 (SCA7)F Hoche, K Seidel, E R Brunt, et al.
Cerebellum (London, England)|December 27, 2011
Pathoanatomy of cerebellar degeneration in spinocerebellar ataxia type 2 (SCA2) and type 3 (SCA3)W Scherzed, E R Brunt, H Heinsen, et al.
Journal of Neural Transmission (Vienna, Austria : 1996)|March 24, 2005
Spinocerebellar ataxias types 2 and 3: degeneration of the pre-cerebellar nuclei isolates the three phylogenetically defined regions of the cerebellumU Rüb, K Gierga, E R Brunt, et al.
Neurology|August 9, 2006
Diffusion tensor MRI shows abnormal brainstem crossing fibers associated with ROBO3 mutationsN L Sicotte, G Salamon, D W Shattuck, et al.
Neuropathology and Applied Neurobiology|February 12, 2009
Spinocerebellar ataxia type 6 (SCA6): neurodegeneration goes beyond the known brain predilection sitesK Gierga, H J Schelhaas, E R Brunt, et al.
Neurology|October 13, 2004
Damage to the reticulotegmental nucleus of the pons in spinocerebellar ataxia type 1, 2, and 3U Rüb, K Bürk, L Schöls, et al.
Neuropathology and Applied Neurobiology|November 1, 2007
Spinocerebellar ataxia type 7 (SCA7): widespread brain damage in an adult-onset patient with progressive visual impairments in comparison with an adult-onset patient without visual impairmentsU Rüb, E R Brunt, K Seidel, et al.
Pageof 8