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Journal of Pharmacological and Toxicological Methods
|
November 1, 1995
SH2 and SH3 domains: potential targets for anti-cancer drug design
T E Smithgall
Pharmacological Reviews
|
April 29, 1998
Signal transduction pathways regulating hematopoietic differentiation
T E Smithgall
The Journal of Biological Chemistry
|
December 20, 1996
Co-expression with BCR induces activation of the FES tyrosine kinase and phosphorylation of specific N-terminal BCR tyrosine residues
J Li, T E Smithgall
The Journal of Biological Chemistry
|
June 5, 1998
Fibroblast transformation by Fps/Fes tyrosine kinases requires Ras, Rac, and Cdc42 and induces extracellular signal-regulated and c-Jun N-terminal kinase activation
J Li, T E Smithgall
Cancer Research
|
October 1, 1985
Sex differences in indomethacin-sensitive 3 alpha-hydroxysteroid dehydrogenase of rat liver cytosol
T E Smithgall, T M Penning
Cancer Research
|
March 8, 2000
The c-Fes protein-tyrosine kinase suppresses cytokine-independent outgrowth of myeloid leukemia cells induced by Bcr-Abl
J M Lionberger, T E Smithgall
Biochemical Pharmacology
|
March 15, 1985
Indomethacin-sensitive 3 alpha-hydroxysteroid dehydrogenase in rat tissues
T E Smithgall, T M Penning
The Journal of Biological Chemistry
|
September 3, 1999
SH2-kinase linker mutations release Hck tyrosine kinase and transforming activities in Rat-2 fibroblasts
S D Briggs, T E Smithgall
Cellular Signalling
|
July 16, 1999
Tyrosine phosphorylation enhances the SH2 domain-binding activity of Bcr and inhibits Bcr interaction with 14-3-3 proteins
K L Peters, T E Smithgall
The Biochemical Journal
|
September 15, 1988
Electrophoretic and immunochemical characterization of 3 alpha-hydroxysteroid/dihydrodiol dehydrogenases of rat tissues
T E Smithgall, T M Penning
Page
of 6
Search research articles
Search
Showing results (1-10 of 58) with videos related to
Sort By:
Page
of 6
Journal of Pharmacological and Toxicological Methods
|
November 1, 1995
SH2 and SH3 domains: potential targets for anti-cancer drug design
T E Smithgall
Pharmacological Reviews
|
April 29, 1998
Signal transduction pathways regulating hematopoietic differentiation
T E Smithgall
The Journal of Biological Chemistry
|
December 20, 1996
Co-expression with BCR induces activation of the FES tyrosine kinase and phosphorylation of specific N-terminal BCR tyrosine residues
J Li, T E Smithgall
The Journal of Biological Chemistry
|
June 5, 1998
Fibroblast transformation by Fps/Fes tyrosine kinases requires Ras, Rac, and Cdc42 and induces extracellular signal-regulated and c-Jun N-terminal kinase activation
J Li, T E Smithgall
Cancer Research
|
October 1, 1985
Sex differences in indomethacin-sensitive 3 alpha-hydroxysteroid dehydrogenase of rat liver cytosol
T E Smithgall, T M Penning
Cancer Research
|
March 8, 2000
The c-Fes protein-tyrosine kinase suppresses cytokine-independent outgrowth of myeloid leukemia cells induced by Bcr-Abl
J M Lionberger, T E Smithgall
Biochemical Pharmacology
|
March 15, 1985
Indomethacin-sensitive 3 alpha-hydroxysteroid dehydrogenase in rat tissues
T E Smithgall, T M Penning
The Journal of Biological Chemistry
|
September 3, 1999
SH2-kinase linker mutations release Hck tyrosine kinase and transforming activities in Rat-2 fibroblasts
S D Briggs, T E Smithgall
Cellular Signalling
|
July 16, 1999
Tyrosine phosphorylation enhances the SH2 domain-binding activity of Bcr and inhibits Bcr interaction with 14-3-3 proteins
K L Peters, T E Smithgall
The Biochemical Journal
|
September 15, 1988
Electrophoretic and immunochemical characterization of 3 alpha-hydroxysteroid/dihydrodiol dehydrogenases of rat tissues
T E Smithgall, T M Penning
Page
of 6