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Cell Death and Differentiation|September 15, 2012
Loss of HtrA2/Omi activity in non-neuronal tissues of adult mice causes premature agingS Kang, J-P Louboutin, P Datta, et al.Experimental Cell Research|February 25, 1997
Inhibition of ICE-related proteases (caspases) and nuclear apoptosis by phenylarsine oxideA Takahashi, P J Goldschmidt-Clermont, E S Alnemri, et al.The Journal of Biological Chemistry|October 25, 1996
The Ced-3/interleukin 1beta converting enzyme-like homolog Mch6 and the lamin-cleaving enzyme Mch2alpha are substrates for the apoptotic mediator CPP32S M Srinivasula, T Fernandes-Alnemri, J Zangrilli, et al.Nature|March 10, 2001
A conserved XIAP-interaction motif in caspase-9 and Smac/DIABLO regulates caspase activity and apoptosisS M Srinivasula, R Hegde, A Saleh, et al.Proceedings of the National Academy of Sciences of the United States of America|August 6, 1996
Cleavage of lamin A by Mch2 alpha but not CPP32: multiple interleukin 1 beta-converting enzyme-related proteases with distinct substrate recognition properties are active in apoptosisA Takahashi, E S Alnemri, Y A Lazebnik, et al.Proceedings of the National Academy of Sciences of the United States of America|July 23, 1996
In vitro activation of CPP32 and Mch3 by Mch4, a novel human apoptotic cysteine protease containing two FADD-like domainsT Fernandes-Alnemri, R C Armstrong, J Krebs, et al.The Journal of Biological Chemistry|July 25, 1997
FLAME-1, a novel FADD-like anti-apoptotic molecule that regulates Fas/TNFR1-induced apoptosisS M Srinivasula, M Ahmad, S Ottilie, et al.Proceedings of the National Academy of Sciences of the United States of America|January 31, 2008
Mutations in NALP12 cause hereditary periodic fever syndromesI Jéru, P Duquesnoy, T Fernandes-Alnemri, et al.Pageof 4