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Human Molecular Genetics|November 1, 1994

Isolation and partial characterization of a chloride channel gene which is expressed in kidney and is a candidate for Dent's disease (an X-linked hereditary nephrolithiasis)S E Fisher, G C Black, S E Lloyd, et al.

Kidney International|July 1, 1978

Hemodialysis bone disease: correlation between clinical, histologic, and other findingsF Alvarez-Ude, T G Feest, M K Ward, et al.

Kidney International|January 5, 2000

Tubular proteinuria defined by a study of Dent's (CLCN5 mutation) and other tubular diseasesA G Norden, S J Scheinman, M M Deschodt-Lanckman, et al.

The Journal of Clinical Investigation|October 6, 1997

Familial distal renal tubular acidosis is associated with mutations in the red cell anion exchanger (Band 3, AE1) geneL J Bruce, D L Cope, G K Jones, et al.

Proceedings of the National Academy of Sciences of the United States of America|August 15, 1992

Apolipoprotein AI mutation Arg-60 causes autosomal dominant amyloidosisA K Soutar, P N Hawkins, D M Vigushin, et al.

Kidney International|November 13, 2001

Glomerular protein sieving and implications for renal failure in Fanconi syndromeA G Norden, M Lapsley, P J Lee, et al.

Journal of Bone and Mineral Research : the Official Journal of the American Society for Bone and Mineral Research|September 1, 1999

Renal chloride channel, CLCN5, mutations in Dent's diseaseJ P Cox, K Yamamoto, P T Christie, et al.

Human Molecular Genetics|August 1, 1997

Characterisation of renal chloride channel, CLCN5, mutations in hypercalciuric nephrolithiasis (kidney stones) disordersS E Lloyd, W Gunther, S H Pearce, et al.

Kidney International|January 5, 2000

Isolated hypercalciuria with mutation in CLCN5: relevance to idiopathic hypercalciuriaS J Scheinman, J P Cox, S E Lloyd, et al.

QJM : Monthly Journal of the Association of Physicians|August 25, 2012

Tropical distal renal tubular acidosis: clinical and epidemiological studies in 78 patientsS Khositseth, L J Bruce, S B Walsh, et al.

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Showing results (31-40 of 43) with videos related to

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Human Molecular Genetics|November 1, 1994

Isolation and partial characterization of a chloride channel gene which is expressed in kidney and is a candidate for Dent's disease (an X-linked hereditary nephrolithiasis)S E Fisher, G C Black, S E Lloyd, et al.

Kidney International|July 1, 1978

Hemodialysis bone disease: correlation between clinical, histologic, and other findingsF Alvarez-Ude, T G Feest, M K Ward, et al.

Kidney International|January 5, 2000

Tubular proteinuria defined by a study of Dent's (CLCN5 mutation) and other tubular diseasesA G Norden, S J Scheinman, M M Deschodt-Lanckman, et al.

The Journal of Clinical Investigation|October 6, 1997

Familial distal renal tubular acidosis is associated with mutations in the red cell anion exchanger (Band 3, AE1) geneL J Bruce, D L Cope, G K Jones, et al.

Proceedings of the National Academy of Sciences of the United States of America|August 15, 1992

Apolipoprotein AI mutation Arg-60 causes autosomal dominant amyloidosisA K Soutar, P N Hawkins, D M Vigushin, et al.

Kidney International|November 13, 2001

Glomerular protein sieving and implications for renal failure in Fanconi syndromeA G Norden, M Lapsley, P J Lee, et al.

Journal of Bone and Mineral Research : the Official Journal of the American Society for Bone and Mineral Research|September 1, 1999

Renal chloride channel, CLCN5, mutations in Dent's diseaseJ P Cox, K Yamamoto, P T Christie, et al.

Human Molecular Genetics|August 1, 1997

Characterisation of renal chloride channel, CLCN5, mutations in hypercalciuric nephrolithiasis (kidney stones) disordersS E Lloyd, W Gunther, S H Pearce, et al.

Kidney International|January 5, 2000

Isolated hypercalciuria with mutation in CLCN5: relevance to idiopathic hypercalciuriaS J Scheinman, J P Cox, S E Lloyd, et al.

QJM : Monthly Journal of the Association of Physicians|August 25, 2012

Tropical distal renal tubular acidosis: clinical and epidemiological studies in 78 patientsS Khositseth, L J Bruce, S B Walsh, et al.

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