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T G Ohm

Showing results (41-50 of 54) with videos related to

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Neuroscience|January 18, 2005
Pathological cholesterol metabolism fails to modify electrophysiological properties of afflicted neurones in Niemann-Pick disease type CR A Deisz, V Meske, S Treiber-Held, et al.
Neuroscience|June 1, 1995
Apolipoprotein E polymorphism influences not only cerebral senile plaque load but also Alzheimer-type neurofibrillary tangle formationT G Ohm, M Kirca, J Bohl, et al.
International Journal of Developmental Neuroscience : the Official Journal of the International Society for Developmental Neuroscience|July 25, 2007
Beta/A4-Amyloid increases nerve growth factor production in rat primary hippocampal astrocyte culturesO Schulte-Herbrüggen, U Hamker, V Meske, et al.
Brain Research|January 12, 2000
Quantitative autoradiography of [3H]forskolin binding sites in post-mortem brain staged for Alzheimer's disease neurofibrillary changes and amyloid depositsA García-Jiménez, R F Cowburn, T G Ohm, et al.
Hippocampus|January 1, 1993
Cellular and molecular pathology in Alzheimer's diseaseK A Crutcher, B H Anderton, S W Barger, et al.
Pharmacopsychiatry|October 24, 2003
Cholesterol and tau protein--findings in Alzheimer's and Niemann Pick C's diseaseT G Ohm, S Treiber-Held, R Distl, et al.
Neurology|June 27, 2001
Gender and age modify the association between APOE and AD-related neuropathologyE Ghebremedhin, C Schultz, D R Thal, et al.
Brain Research|August 5, 1998
Loss of inositol 1,4,5-trisphosphate receptor sites and decreased PKC levels correlate with staging of Alzheimer's disease neurofibrillary pathologyT Kurumatani, J Fastbom, W L Bonkale, et al.
Journal of Neuroscience Research|January 29, 2002
Loss of stimulatory effect of guanosine triphosphate on [(35)S]GTPgammaS binding correlates with Alzheimer's disease neurofibrillary pathology in entorhinal cortex and CA1 hippocampal subfieldA García-Jiménez, R F Cowburn, T G Ohm, et al.
Biochemical Society Symposium|July 13, 2001
Dysfunctional intracellular calcium homoeostasis: a central cause of neurodegeneration in Alzheimer's diseaseC O'Neill, R F Cowburn, W L Bonkale, et al.
Pageof 6

Showing results (41-50 of 54) with videos related to

Sort By:
Pageof 6
Neuroscience|January 18, 2005
Pathological cholesterol metabolism fails to modify electrophysiological properties of afflicted neurones in Niemann-Pick disease type CR A Deisz, V Meske, S Treiber-Held, et al.
Neuroscience|June 1, 1995
Apolipoprotein E polymorphism influences not only cerebral senile plaque load but also Alzheimer-type neurofibrillary tangle formationT G Ohm, M Kirca, J Bohl, et al.
International Journal of Developmental Neuroscience : the Official Journal of the International Society for Developmental Neuroscience|July 25, 2007
Beta/A4-Amyloid increases nerve growth factor production in rat primary hippocampal astrocyte culturesO Schulte-Herbrüggen, U Hamker, V Meske, et al.
Brain Research|January 12, 2000
Quantitative autoradiography of [3H]forskolin binding sites in post-mortem brain staged for Alzheimer's disease neurofibrillary changes and amyloid depositsA García-Jiménez, R F Cowburn, T G Ohm, et al.
Hippocampus|January 1, 1993
Cellular and molecular pathology in Alzheimer's diseaseK A Crutcher, B H Anderton, S W Barger, et al.
Pharmacopsychiatry|October 24, 2003
Cholesterol and tau protein--findings in Alzheimer's and Niemann Pick C's diseaseT G Ohm, S Treiber-Held, R Distl, et al.
Neurology|June 27, 2001
Gender and age modify the association between APOE and AD-related neuropathologyE Ghebremedhin, C Schultz, D R Thal, et al.
Brain Research|August 5, 1998
Loss of inositol 1,4,5-trisphosphate receptor sites and decreased PKC levels correlate with staging of Alzheimer's disease neurofibrillary pathologyT Kurumatani, J Fastbom, W L Bonkale, et al.
Journal of Neuroscience Research|January 29, 2002
Loss of stimulatory effect of guanosine triphosphate on [(35)S]GTPgammaS binding correlates with Alzheimer's disease neurofibrillary pathology in entorhinal cortex and CA1 hippocampal subfieldA García-Jiménez, R F Cowburn, T G Ohm, et al.
Biochemical Society Symposium|July 13, 2001
Dysfunctional intracellular calcium homoeostasis: a central cause of neurodegeneration in Alzheimer's diseaseC O'Neill, R F Cowburn, W L Bonkale, et al.
Pageof 6