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Genome|February 1, 1993
Quantitative trait loci on barley (Hordeum vulgare L.) chromosome 7 associated with components of winterhardinessP M Hayes, T Blake, T H Chen, et al.
Biometrics|April 28, 2000
Estimation of sojourn time in chronic disease screening without data on interval casesT H Chen, H S Kuo, M F Yen, et al.
American Journal of Physiology. Endocrinology and Metabolism|June 19, 2001
Parathyroid cells express dihydropyridine-sensitive cation currents and L-type calcium channel subunitsW Chang, S A Pratt, T H Chen, et al.
Experimental and Clinical Endocrinology & Diabetes : Official Journal, German Society of Endocrinology [And] German Diabetes Association|October 17, 2012
Mental health status and factors that influence the course of Graves' disease and antithyroid treatmentsD Y Chen, P F Schneider, X S Zhang, et al.
Plant Molecular Biology|July 21, 1999
Enhanced tolerance to light stress of transgenic Arabidopsis plants that express the codA gene for a bacterial choline oxidaseAlia, Y Kondo, A Sakamoto, et al.
Zhonghua Yi Xue Za Zhi = Chinese Medical Journal; Free China Ed|October 5, 2001
The in vitro inhibitory effect of flavonoid astilbin on 3-hydroxy-3-methylglutaryl coenzyme A reductase on Vero cellsT H Chen, J C Liu, J J Chang, et al.
Plant Physiology|July 1, 1984
Cryopreservation of Alkaloid-Producing Cell Cultures of Periwinkle (Catharanthus roseus)T H Chen, K K Kartha, N L Leung, et al.
The Journal of Biological Chemistry|December 27, 1996
The N-terminal region of the third intracellular loop of the parathyroid hormone (PTH)/PTH-related peptide receptor is critical for coupling to cAMP and inositol phosphate/Ca2+ signal transduction pathwaysZ Huang, Y Chen, S Pratt, et al.
Molecular Endocrinology (Baltimore, Md.)|September 1, 1995
Mutational analysis of the cytoplasmic tail of the G protein-coupled receptor for parathyroid hormone (PTH) and PTH-related protein: effects on receptor expression and signalingZ Huang, Y Chen, S Pratt, et al.
American Journal of Human Genetics|January 1, 1992
Mucolipidoses II and III variants with normal N-acetylglucosamine 1-phosphotransferase activity toward alpha-methylmannoside are due to nonallelic mutationsY Ben-Yoseph, D A Mitchell, R M Yager, et al.
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