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American Journal of Medical Genetics|September 19, 1997

Ehlers-Danlos syndrome type VIIA and VIIB result from splice-junction mutations or genomic deletions that involve exon 6 in the COL1A1 and COL1A2 genes of type I collagenP H Byers, M Duvic, M Atkinson, et al.

The Journal of Cell Biology|February 28, 1998

Mice that lack thrombospondin 2 display connective tissue abnormalities that are associated with disordered collagen fibrillogenesis, an increased vascular density, and a bleeding diathesisT R Kyriakides, Y H Zhu, L T Smith, et al.

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Showing results (171-180 of 172) with videos related to

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American Journal of Medical Genetics|September 19, 1997

Ehlers-Danlos syndrome type VIIA and VIIB result from splice-junction mutations or genomic deletions that involve exon 6 in the COL1A1 and COL1A2 genes of type I collagenP H Byers, M Duvic, M Atkinson, et al.

The Journal of Cell Biology|February 28, 1998

Mice that lack thrombospondin 2 display connective tissue abnormalities that are associated with disordered collagen fibrillogenesis, an increased vascular density, and a bleeding diathesisT R Kyriakides, Y H Zhu, L T Smith, et al.

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