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T Hofer

Showing results (51-60 of 66) with videos related to

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Molecular Autism|January 11, 2020
Biophysical classification of a <i>CACNA1D</i> de novo mutation as a high-risk mutation for a severe neurodevelopmental disorderNadja T Hofer, Petronel Tuluc, Nadine J Ortner, et al.
The Journal of Physiology|November 28, 2017
Hyperexcitability of the network contributes to synchronization processes in the human epileptic neocortexKinga Tóth, Katharina T Hofer, Ágnes Kandrács, et al.
Scientific Reports|April 16, 2022
Bursting of excitatory cells is linked to interictal epileptic discharge generation in humansKatharina T Hofer, Ágnes Kandrács, Kinga Tóth, et al.
The Journal of Physiology|September 17, 2019
Presence of synchrony-generating hubs in the human epileptic neocortexÁgnes Kandrács, Katharina T Hofer, Kinga Tóth, et al.
Andrology|August 31, 2016
Enhanced susceptibility of obese mice to glycidamide-induced sperm chromatin damage without increased oxidative stressK B Gutzkow, N Duale, T Danielsen, et al.
Pflugers Archiv : European Journal of Physiology|December 19, 2019
RBP2 stabilizes slow Cav1.3 Ca<sup>2+</sup> channel inactivation properties of cochlear inner hair cellsNadine J Ortner, Alexandra Pinggera, Nadja T Hofer, et al.
Materials Science & Engineering. C, Materials for Biological Applications|May 16, 2020
The neural tissue around SU-8 implants: A quantitative in vivo biocompatibility studyGergely Márton, Estilla Zsófia Tóth, Lucia Wittner, et al.
The Journal of Physiology|January 11, 2019
Impaired chromaffin cell excitability and exocytosis in autistic Timothy syndrome TS2-neo mouse rescued by L-type calcium channel blockersChiara Calorio, Daniela Gavello, Laura Guarina, et al.
Elife|July 6, 2022
β2-subunit alternative splicing stabilizes Cav2.3 Ca<sup>2+</sup> channel activity during continuous midbrain dopamine neuron-like activityAnita Siller, Nadja T Hofer, Giulia Tomagra, et al.
Science (New York, N.Y.)|July 16, 2005
Mitochondrial DNA mutations, oxidative stress, and apoptosis in mammalian agingG C Kujoth, A Hiona, T D Pugh, et al.
Pageof 7

Showing results (51-60 of 66) with videos related to

Sort By:
Pageof 7
Molecular Autism|January 11, 2020
Biophysical classification of a <i>CACNA1D</i> de novo mutation as a high-risk mutation for a severe neurodevelopmental disorderNadja T Hofer, Petronel Tuluc, Nadine J Ortner, et al.
The Journal of Physiology|November 28, 2017
Hyperexcitability of the network contributes to synchronization processes in the human epileptic neocortexKinga Tóth, Katharina T Hofer, Ágnes Kandrács, et al.
Scientific Reports|April 16, 2022
Bursting of excitatory cells is linked to interictal epileptic discharge generation in humansKatharina T Hofer, Ágnes Kandrács, Kinga Tóth, et al.
The Journal of Physiology|September 17, 2019
Presence of synchrony-generating hubs in the human epileptic neocortexÁgnes Kandrács, Katharina T Hofer, Kinga Tóth, et al.
Andrology|August 31, 2016
Enhanced susceptibility of obese mice to glycidamide-induced sperm chromatin damage without increased oxidative stressK B Gutzkow, N Duale, T Danielsen, et al.
Pflugers Archiv : European Journal of Physiology|December 19, 2019
RBP2 stabilizes slow Cav1.3 Ca<sup>2+</sup> channel inactivation properties of cochlear inner hair cellsNadine J Ortner, Alexandra Pinggera, Nadja T Hofer, et al.
Materials Science & Engineering. C, Materials for Biological Applications|May 16, 2020
The neural tissue around SU-8 implants: A quantitative in vivo biocompatibility studyGergely Márton, Estilla Zsófia Tóth, Lucia Wittner, et al.
The Journal of Physiology|January 11, 2019
Impaired chromaffin cell excitability and exocytosis in autistic Timothy syndrome TS2-neo mouse rescued by L-type calcium channel blockersChiara Calorio, Daniela Gavello, Laura Guarina, et al.
Elife|July 6, 2022
β2-subunit alternative splicing stabilizes Cav2.3 Ca<sup>2+</sup> channel activity during continuous midbrain dopamine neuron-like activityAnita Siller, Nadja T Hofer, Giulia Tomagra, et al.
Science (New York, N.Y.)|July 16, 2005
Mitochondrial DNA mutations, oxidative stress, and apoptosis in mammalian agingG C Kujoth, A Hiona, T D Pugh, et al.
Pageof 7