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The Biochemical Journal
|
April 1, 1989
Influx of bivalent cations can be independent of receptor stimulation in human endothelial cells
T J Hallam, R Jacob, J E Merritt
The Biochemical Journal
|
October 1, 1988
Evidence that agonists stimulate bivalent-cation influx into human endothelial cells
T J Hallam, R Jacob, J E Merritt
The Biochemical Journal
|
March 15, 1984
Stimulus-response coupling in human platelets. Changes evoked by platelet-activating factor in cytoplasmic free calcium monitored with the fluorescent calcium indicator quin2
T J Hallam, A Sanchez, T J Rink
FEBS Letters
|
November 28, 1983
Trifluoperazine and chlorpromazine block secretion from human platelets evoked at basal cytoplasmic free calcium by activators of C-kinase
A Sanchez, T J Hallam, T J Rink
The Journal of Biological Chemistry
|
January 25, 1989
Use of manganese to discriminate between calcium influx and mobilization from internal stores in stimulated human neutrophils
J E Merritt, R Jacob, T J Hallam
Nature
|
September 22, 1983
Diacylglycerol and phorbol ester stimulate secretion without raising cytoplasmic free calcium in human platelets
T J Rink, A Sanchez, T J Hallam
The Biochemical Journal
|
April 1, 1990
A role for calcium and protein kinase C in agonist-stimulated adhesion of human neutrophils
M P Davies, T J Hallam, J E Merritt
FEBS Letters
|
October 3, 1983
The role of 1-O-alkyl-2-acetyl-sn-glyceryl-3-phosphorylcholine (AcGEPC) and palmitoyl-lysophosphatidate in the responses of human blood platelets to collagen and thrombin
T J Hallam, M C Scrutton, R B Wallis
Biochemical Society Transactions
|
November 1, 1993
The role of protein kinase C in TCR-induced interleukin-2 secretion in human Jurkat T cells
D H Williams, T J Hallam, E J Murray
The Biochemical Journal
|
December 1, 1989
Activation of protein kinase C in human neutrophils attenuates agonist-stimulated rises in cytosolic free Ca2+ concentration by inhibiting bivalent-cation influx and intracellular Ca2+ release in addition to stimulating Ca2+ efflux
S A McCarthy, T J Hallam, J E Merritt
Page
of 5
Search research articles
Search
Showing results (11-20 of 49) with videos related to
Sort By:
Page
of 5
The Biochemical Journal
|
April 1, 1989
Influx of bivalent cations can be independent of receptor stimulation in human endothelial cells
T J Hallam, R Jacob, J E Merritt
The Biochemical Journal
|
October 1, 1988
Evidence that agonists stimulate bivalent-cation influx into human endothelial cells
T J Hallam, R Jacob, J E Merritt
The Biochemical Journal
|
March 15, 1984
Stimulus-response coupling in human platelets. Changes evoked by platelet-activating factor in cytoplasmic free calcium monitored with the fluorescent calcium indicator quin2
T J Hallam, A Sanchez, T J Rink
FEBS Letters
|
November 28, 1983
Trifluoperazine and chlorpromazine block secretion from human platelets evoked at basal cytoplasmic free calcium by activators of C-kinase
A Sanchez, T J Hallam, T J Rink
The Journal of Biological Chemistry
|
January 25, 1989
Use of manganese to discriminate between calcium influx and mobilization from internal stores in stimulated human neutrophils
J E Merritt, R Jacob, T J Hallam
Nature
|
September 22, 1983
Diacylglycerol and phorbol ester stimulate secretion without raising cytoplasmic free calcium in human platelets
T J Rink, A Sanchez, T J Hallam
The Biochemical Journal
|
April 1, 1990
A role for calcium and protein kinase C in agonist-stimulated adhesion of human neutrophils
M P Davies, T J Hallam, J E Merritt
FEBS Letters
|
October 3, 1983
The role of 1-O-alkyl-2-acetyl-sn-glyceryl-3-phosphorylcholine (AcGEPC) and palmitoyl-lysophosphatidate in the responses of human blood platelets to collagen and thrombin
T J Hallam, M C Scrutton, R B Wallis
Biochemical Society Transactions
|
November 1, 1993
The role of protein kinase C in TCR-induced interleukin-2 secretion in human Jurkat T cells
D H Williams, T J Hallam, E J Murray
The Biochemical Journal
|
December 1, 1989
Activation of protein kinase C in human neutrophils attenuates agonist-stimulated rises in cytosolic free Ca2+ concentration by inhibiting bivalent-cation influx and intracellular Ca2+ release in addition to stimulating Ca2+ efflux
S A McCarthy, T J Hallam, J E Merritt
Page
of 5