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Journal of Orthopaedic Research : Official Publication of the Orthopaedic Research Society|April 4, 2006
Cartilage tissue engineering by expanded goat articular chondrocytesS Miot, P Scandiucci de Freitas, D Wirz, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|October 24, 2008
NaV1.7 gain-of-function mutations as a continuum: A1632E displays physiological changes associated with erythromelalgia and paroxysmal extreme pain disorder mutations and produces symptoms of both disordersM Estacion, S D Dib-Hajj, P J Benke, et al.Oral Microbiology and Immunology|February 1, 1997
Recognition of antigenic epitopes in lipopolysaccharide and protein from Actinobacillus actinomycetemcomitans by serum antibodies in untreated rapidly progressive periodontitis patientsJ G Ou, B Bainbridge, K Gu, et al.European Journal of Clinical Investigation|February 1, 1980
Biochemistry and histology of the connective tissue of Dupuytren's disease lesionsS Bazin, M Le Lous, V C Duance, et al.Neurobiology of Disease|March 25, 2014
Physiological and genetic analysis of multiple sodium channel variants in a model of genetic absence epilepsyM K Oliva, T C McGarr, B J Beyer, et al.Spinal Cord|December 21, 2011
International spinal cord injury pain classification: part I. Background and description. March 6-7, 2009T N Bryce, F Biering-Sørensen, N B Finnerup, et al.Spinal Cord|February 8, 2012
International Spinal Cord Injury Pain (ISCIP) Classification: Part 2. Initial validation using vignettesT N Bryce, F Biering-Sørensen, N B Finnerup, et al.Neurology|April 28, 2012
Nav1.7-related small fiber neuropathy: impaired slow-inactivation and DRG neuron hyperexcitabilityC Han, J G J Hoeijmakers, H-S Ahn, et al.Archives of Neurology|September 15, 2005
Characterizing the mechanisms of progression in multiple sclerosis: evidence and new hypotheses for future directionsE M Frohman, M Filippi, O Stuve, et al.Pageof 41