Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

T Jacks

Showing results (61-70 of 130) with videos related to

Pageof 13
Sort By:
Current Biology : CB|July 1, 1993
Generation of normal lymphocyte populations by Rb-deficient embryonic stem cellsJ Chen, J R Gorman, V Stewart, et al.
Cell|September 18, 1992
Altered cell cycle arrest and gene amplification potential accompany loss of wild-type p53L R Livingstone, A White, J Sprouse, et al.
Cold Spring Harbor Symposia on Quantitative Biology|January 1, 1994
p53-dependent apoptosis in vivo: impact of p53 inactivation on tumorigenesisH Symonds, L Krall, L Remington, et al.
Nature Genetics|April 16, 1998
Loss of E2F-1 reduces tumorigenesis and extends the lifespan of Rb1(+/-)miceL Yamasaki, R Bronson, B O Williams, et al.
Nature Genetics|September 6, 2000
Nf1;Trp53 mutant mice develop glioblastoma with evidence of strain-specific effectsK M Reilly, D A Loisel, R T Bronson, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 3, 1999
Inhibition of cyclin-dependent kinase 2 by p21 is necessary for retinoblastoma protein-mediated G1 arrest after gamma-irradiationJ Brugarolas, K Moberg, S D Boyd, et al.
Oncogene|March 21, 1996
Identification and cloning of EI24, a gene induced by p53 in etoposide-treated cellsS M Lehar, M Nacht, T Jacks, et al.
Genes & Development|April 1, 1993
Oct-2, although not required for early B-cell development, is critical for later B-cell maturation and for postnatal survivalL M Corcoran, M Karvelas, G J Nossal, et al.
Nature Genetics|August 1, 1994
Cooperative tumorigenic effects of germline mutations in Rb and p53B O Williams, L Remington, D M Albert, et al.
Proceedings of the National Academy of Sciences of the United States of America|October 1, 1993
Cell proliferation, DNA repair, and p53 function are not required for programmed death of prostatic glandular cells induced by androgen ablationR R Berges, Y Furuya, L Remington, et al.
Pageof 13

Showing results (61-70 of 130) with videos related to

Sort By:
Pageof 13
Current Biology : CB|July 1, 1993
Generation of normal lymphocyte populations by Rb-deficient embryonic stem cellsJ Chen, J R Gorman, V Stewart, et al.
Cell|September 18, 1992
Altered cell cycle arrest and gene amplification potential accompany loss of wild-type p53L R Livingstone, A White, J Sprouse, et al.
Cold Spring Harbor Symposia on Quantitative Biology|January 1, 1994
p53-dependent apoptosis in vivo: impact of p53 inactivation on tumorigenesisH Symonds, L Krall, L Remington, et al.
Nature Genetics|April 16, 1998
Loss of E2F-1 reduces tumorigenesis and extends the lifespan of Rb1(+/-)miceL Yamasaki, R Bronson, B O Williams, et al.
Nature Genetics|September 6, 2000
Nf1;Trp53 mutant mice develop glioblastoma with evidence of strain-specific effectsK M Reilly, D A Loisel, R T Bronson, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 3, 1999
Inhibition of cyclin-dependent kinase 2 by p21 is necessary for retinoblastoma protein-mediated G1 arrest after gamma-irradiationJ Brugarolas, K Moberg, S D Boyd, et al.
Oncogene|March 21, 1996
Identification and cloning of EI24, a gene induced by p53 in etoposide-treated cellsS M Lehar, M Nacht, T Jacks, et al.
Genes & Development|April 1, 1993
Oct-2, although not required for early B-cell development, is critical for later B-cell maturation and for postnatal survivalL M Corcoran, M Karvelas, G J Nossal, et al.
Nature Genetics|August 1, 1994
Cooperative tumorigenic effects of germline mutations in Rb and p53B O Williams, L Remington, D M Albert, et al.
Proceedings of the National Academy of Sciences of the United States of America|October 1, 1993
Cell proliferation, DNA repair, and p53 function are not required for programmed death of prostatic glandular cells induced by androgen ablationR R Berges, Y Furuya, L Remington, et al.
Pageof 13