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Immunity|November 5, 1999
Differential roles of TLR2 and TLR4 in recognition of gram-negative and gram-positive bacterial cell wall componentsO Takeuchi, K Hoshino, T Kawai, et al.
The Journal of Allergy and Clinical Immunology|January 12, 2000
IL-18 deficiency selectively enhances allergen-induced eosinophilia in miceT Kodama, T Matsuyama, K Kuribayashi, et al.
Genes to Cells : Devoted to Molecular & Cellular Mechanisms|March 30, 1999
Targeted disruption of ATF4 discloses its essential role in the formation of eye lens fibresT Tanaka, T Tsujimura, K Takeda, et al.
European Journal of Biochemistry|July 15, 1995
Identification of a transcriptional regulatory factor for human aromatase cytochrome P450 gene expression as nuclear factor interleukin-6 (NF-IL6), a member of the CCAAT/enhancer-binding protein familyK Toda, S Akira, T Kishimoto, et al.
The Journal of Experimental Medicine|June 6, 1998
Abrogation of bronchial eosinophilic inflammation and airway hyperreactivity in signal transducers and activators of transcription (STAT)6-deficient miceT Akimoto, F Numata, M Tamura, et al.
The New Biologist|January 1, 1991
Reciprocal expression of NF-IL6 and C/EBP in hepatocytes: possible involvement of NF-IL6 in acute phase protein gene expressionH Isshiki, S Akira, T Sugita, et al.
Immunity|June 8, 2000
Caspase-1 activation of IL-1beta and IL-18 are essential for Shigella flexneri-induced inflammationP J Sansonetti, A Phalipon, J Arondel, et al.
Oncogene|June 22, 1999
Death-associated protein kinase 2 is a new calcium/calmodulin-dependent protein kinase that signals apoptosis through its catalytic activityT Kawai, F Nomura, K Hoshino, et al.
Journal of Immunology (Baltimore, Md. : 1950)|January 7, 2000
Cutting edge: preferentially the R-stereoisomer of the mycoplasmal lipopeptide macrophage-activating lipopeptide-2 activates immune cells through a toll-like receptor 2- and MyD88-dependent signaling pathwayO Takeuchi, A Kaufmann, K Grote, et al.
Proceedings of the National Academy of Sciences of the United States of America|November 26, 1999
IL-18, although antiallergic when administered with IL-12, stimulates IL-4 and histamine release by basophilsT Yoshimoto, H Tsutsui, K Tominaga, et al.
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