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The American Journal of Physiology|July 1, 1996
cGMP and cAMP in prostaglandin-induced pial artery dilation and increased CSF opioid concentrationW M ArmsteadJournal of Neurotrauma|November 1, 2001
Endothelin-Induced cyclooxygenase-dependent superoxide generation contributes to K+ channel functional impairment after brain injuryW M ArmsteadThe American Journal of Physiology|September 2, 1998
Role of opioids in hypoxic pial artery dilation is stimulus duration dependentW M ArmsteadBrain Research|August 8, 2001
Vasopressin induced cyclooxygenase dependent superoxide generation contributes to K(+) channel function impairment after brain injuryW M ArmsteadBrain Research|April 27, 1999
Stimulus duration modulates the interaction between opioids and nitric oxide in hypoxic pial artery dilationW M ArmsteadJournal of Neurotrauma|September 30, 2000
Role of nociceptin/orphanin FQ in age-dependent cerebral hemodynamic effects of brain injuryW M ArmsteadMicrocirculation (New York, N.Y. : 1994)|August 30, 2000
Age-dependent cerebral hemodynamic effects of traumatic brain injury in newborn and juvenile pigsW M ArmsteadBrain Research|August 21, 1995
Relationship between opioids and activation of phospholipase C and protein kinase C in brain injury induced pial artery vasoconstrictionW M ArmsteadBrain Research|July 17, 1998
Contribution of kca channel activation to hypoxic cerebrovasodilation does not involve NOW M ArmsteadCurrent Pharmaceutical Design|July 30, 2004
Endothelins and the role of endothelin antagonists in the management of posttraumatic vasospasmW M ArmsteadPageof 13