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The American Journal of Physiology|June 5, 1998
ATP-dependent K+ channel activation reduces loss of opioid dilation after brain injuryW M ArmsteadStroke|November 22, 1997
Brain injury impairs ATP-sensitive K+ channel function in piglet cerebral arteriesW M ArmsteadBrain Research|November 22, 1997
Role of impaired cAMP and calcium-sensitive K+ channel function in altered cerebral hemodynamics following brain injuryW M ArmsteadBrain Research|March 18, 2000
Altered release of prostaglandins contributes to hypoxic/ischemic impairment of NOC/oFQ cerebrovasodilationW M ArmsteadAmerican Journal of Physiology. Heart and Circulatory Physiology|October 25, 2000
NOC/oFQ contributes to age-dependent impairment of NMDA-induced cerebrovasodilation after brain injuryW M ArmsteadBrain Research|June 24, 2000
Role of NOC/oFQ in impaired opioid-induced pial artery dilation following brain injuryW M ArmsteadPeptides|February 17, 2001
Age dependent endothelin contribution to NOC/oFQ induced impairment of NMDA cerebrovasodilation after brain injuryW M ArmsteadAmerican Journal of Physiology. Heart and Circulatory Physiology|February 9, 2000
Relationship between nociceptin/orphanin FQ and cerebral hemodynamics after hypoxia-ischemia in pigletsW M ArmsteadThe American Journal of Physiology|February 1, 1996
Role of ATP-sensitive K+ channels in cGMP-mediated pial artery vasodilationW M ArmsteadProceedings of the Society for Experimental Biology and Medicine. Society for Experimental Biology and Medicine (New York, N.Y.)|June 1, 1996
Relationship between opioids and prostaglandins in hypoxia-induced vasodilation of pial arteries in the newborn pigW M ArmsteadPageof 13