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The American Journal of Physiology|April 1, 1995
Role of nitric oxide and cAMP in prostaglandin-induced pial arterial vasodilationW M ArmsteadJournal of Cerebral Blood Flow and Metabolism : Official Journal of the International Society of Cerebral Blood Flow and Metabolism|January 1, 1997
Role of nitric oxide, cyclic nucleotides, and the activation of ATP-sensitive K+ channels in the contribution of adenosine to hypoxia-induced pial artery dilationW M ArmsteadBrain Research|December 5, 1998
Nitric oxide contributes to opioid release from glia during hypoxiaW M ArmsteadThe American Journal of Physiology|April 1, 1997
Role of activation of calcium-sensitive K+ channels in NO- and hypoxia-induced pial artery vasodilationW M ArmsteadJournal of Neurotrauma|September 30, 1998
Brain injury impairs prostaglandin cerebrovasodilationW M ArmsteadBrain Research|February 7, 1997
Role of activation of calcium-sensitive K+ channels and cAMP in opioid-induced pial artery dilationW M ArmsteadBrain Research|June 8, 2000
NOC/oFQ contributes to hypoxic-ischemic impairment of N-methyl-D-aspartate-induced cerebral vasodilationW M ArmsteadBrain Research|October 20, 1999
Endothelin-1 contributes to normocapnic hyperoxic pial artery vasoconstrictionW M ArmsteadThe American Journal of Physiology|November 24, 1999
Role of endothelin-1 in age-dependent cerebrovascular hypotensive responses after brain injuryW M ArmsteadBrain Research Bulletin|February 17, 2001
Role of altered cyclooxygenase metabolism in impaired cerebrovasodilation to nociceptin/orphanin FQ following brain injuryW M ArmsteadPageof 13