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T Keating

Showing results (101-110 of 213) with videos related to

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Diabetologia|November 13, 2017
Epigenetics in diabetic nephropathy, immunity and metabolismSamuel T Keating, Janna A van Diepen, Niels P Riksen, et al.
Development (Cambridge, England)|October 26, 2002
Mps1 defines a proximal blastemal proliferative compartment essential for zebrafish fin regenerationKenneth D Poss, Alex Nechiporuk, Ann M Hillam, et al.
Developmental Biology|February 15, 2005
Normal newt limb regeneration requires matrix metalloproteinase functionVladimir Vinarsky, Donald L Atkinson, Tamara J Stevenson, et al.
Genomics|May 15, 1997
Genomic localization of the human gene for KCNA10, a cGMP-activated K channelM Orias, P Bray-Ward, M E Curran, et al.
The New England Journal of Medicine|May 29, 1997
Molecular basis of the long-QT syndrome associated with deafnessI Splawski, K W Timothy, G M Vincent, et al.
Nature Genetics|November 14, 1997
Mutations in the hminK gene cause long QT syndrome and suppress IKs functionI Splawski, M Tristani-Firouzi, M H Lehmann, et al.
Proceedings of the Association of American Physicians|September 1, 1997
George M. Cober Lecturer: Mark T. Keating. Molecular basis of the long-QT syndrome associated with deafnessI Splawski, K W Timothy, G M Vincent, et al.
The Journal of General Physiology|May 1, 1996
Fast inactivation causes rectification of the IKr channelP S Spector, M E Curran, A Zou, et al.
Developmental Dynamics : an Official Publication of the American Association of Anatomists|December 24, 2005
Tissue inhibitor of metalloproteinase 1 regulates matrix metalloproteinase activity during newt limb regenerationTamara J Stevenson, Vladimir Vinarsky, Donald L Atkinson, et al.
The Journal of Biological Chemistry|July 20, 1999
Long QT syndrome-associated mutations in the S4-S5 linker of KvLQT1 potassium channels modify gating and interaction with minK subunitsL Franqueza, M Lin, J Shen, et al.
Pageof 22

Showing results (101-110 of 213) with videos related to

Sort By:
Pageof 22
Diabetologia|November 13, 2017
Epigenetics in diabetic nephropathy, immunity and metabolismSamuel T Keating, Janna A van Diepen, Niels P Riksen, et al.
Development (Cambridge, England)|October 26, 2002
Mps1 defines a proximal blastemal proliferative compartment essential for zebrafish fin regenerationKenneth D Poss, Alex Nechiporuk, Ann M Hillam, et al.
Developmental Biology|February 15, 2005
Normal newt limb regeneration requires matrix metalloproteinase functionVladimir Vinarsky, Donald L Atkinson, Tamara J Stevenson, et al.
Genomics|May 15, 1997
Genomic localization of the human gene for KCNA10, a cGMP-activated K channelM Orias, P Bray-Ward, M E Curran, et al.
The New England Journal of Medicine|May 29, 1997
Molecular basis of the long-QT syndrome associated with deafnessI Splawski, K W Timothy, G M Vincent, et al.
Nature Genetics|November 14, 1997
Mutations in the hminK gene cause long QT syndrome and suppress IKs functionI Splawski, M Tristani-Firouzi, M H Lehmann, et al.
Proceedings of the Association of American Physicians|September 1, 1997
George M. Cober Lecturer: Mark T. Keating. Molecular basis of the long-QT syndrome associated with deafnessI Splawski, K W Timothy, G M Vincent, et al.
The Journal of General Physiology|May 1, 1996
Fast inactivation causes rectification of the IKr channelP S Spector, M E Curran, A Zou, et al.
Developmental Dynamics : an Official Publication of the American Association of Anatomists|December 24, 2005
Tissue inhibitor of metalloproteinase 1 regulates matrix metalloproteinase activity during newt limb regenerationTamara J Stevenson, Vladimir Vinarsky, Donald L Atkinson, et al.
The Journal of Biological Chemistry|July 20, 1999
Long QT syndrome-associated mutations in the S4-S5 linker of KvLQT1 potassium channels modify gating and interaction with minK subunitsL Franqueza, M Lin, J Shen, et al.
Pageof 22