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T Kossmann

Showing results (71-80 of 78) with videos related to

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Journal of Cerebral Blood Flow and Metabolism : Official Journal of the International Society of Cerebral Blood Flow and Metabolism|March 1, 1997
Interleukin-8 released into the cerebrospinal fluid after brain injury is associated with blood-brain barrier dysfunction and nerve growth factor productionT Kossmann, P F Stahel, P M Lenzlinger, et al.
Growth Factors (Chur, Switzerland)|January 1, 1992
Kupffer cells express type I TGF-beta receptors, migrate to TGF-beta and participate in streptococcal cell wall induced hepatic granuloma formationT Kossmann, C L Manthey, M E Brandes, et al.
Neuroreport|July 4, 2001
Regulation of chemokines and chemokine receptors after experimental closed head injuryV I Otto, P F Stahel, M Rancan, et al.
The Journal of Experimental Medicine|April 1, 1991
Macrophage- and astrocyte-derived transforming growth factor beta as a mediator of central nervous system dysfunction in acquired immune deficiency syndromeS M Wahl, J B Allen, N McCartney-Francis, et al.
Injury|January 18, 2006
Characterisation of orthopaedic trauma admitted to adult level 1 trauma centresD M Urquhart, E R Edwards, S E Graves, et al.
Journal of Cerebral Blood Flow and Metabolism : Official Journal of the International Society of Cerebral Blood Flow and Metabolism|March 4, 2000
Experimental closed head injury: analysis of neurological outcome, blood-brain barrier dysfunction, intracranial neutrophil infiltration, and neuronal cell death in mice deficient in genes for pro-inflammatory cytokinesP F Stahel, E Shohami, F M Younis, et al.
Journal of Neurochemistry|December 1, 1998
Traumatic brain injury increases beta-amyloid peptide 1-42 in cerebrospinal fluidC A Raby, M C Morganti-Kossmann, T Kossmann, et al.
Annals of the New York Academy of Sciences|May 20, 2000
Traumatic brain injury elevates the Alzheimer's amyloid peptide A beta 42 in human CSF. A possible role for nerve cell injuryM R Emmerling, M C Morganti-Kossmann, T Kossmann, et al.
Pageof 8

Showing results (71-80 of 78) with videos related to

Sort By:
Pageof 8
You have reached the last page of results.This site can display upto 78 results.
Journal of Cerebral Blood Flow and Metabolism : Official Journal of the International Society of Cerebral Blood Flow and Metabolism|March 1, 1997
Interleukin-8 released into the cerebrospinal fluid after brain injury is associated with blood-brain barrier dysfunction and nerve growth factor productionT Kossmann, P F Stahel, P M Lenzlinger, et al.
Growth Factors (Chur, Switzerland)|January 1, 1992
Kupffer cells express type I TGF-beta receptors, migrate to TGF-beta and participate in streptococcal cell wall induced hepatic granuloma formationT Kossmann, C L Manthey, M E Brandes, et al.
Neuroreport|July 4, 2001
Regulation of chemokines and chemokine receptors after experimental closed head injuryV I Otto, P F Stahel, M Rancan, et al.
The Journal of Experimental Medicine|April 1, 1991
Macrophage- and astrocyte-derived transforming growth factor beta as a mediator of central nervous system dysfunction in acquired immune deficiency syndromeS M Wahl, J B Allen, N McCartney-Francis, et al.
Injury|January 18, 2006
Characterisation of orthopaedic trauma admitted to adult level 1 trauma centresD M Urquhart, E R Edwards, S E Graves, et al.
Journal of Cerebral Blood Flow and Metabolism : Official Journal of the International Society of Cerebral Blood Flow and Metabolism|March 4, 2000
Experimental closed head injury: analysis of neurological outcome, blood-brain barrier dysfunction, intracranial neutrophil infiltration, and neuronal cell death in mice deficient in genes for pro-inflammatory cytokinesP F Stahel, E Shohami, F M Younis, et al.
Journal of Neurochemistry|December 1, 1998
Traumatic brain injury increases beta-amyloid peptide 1-42 in cerebrospinal fluidC A Raby, M C Morganti-Kossmann, T Kossmann, et al.
Annals of the New York Academy of Sciences|May 20, 2000
Traumatic brain injury elevates the Alzheimer's amyloid peptide A beta 42 in human CSF. A possible role for nerve cell injuryM R Emmerling, M C Morganti-Kossmann, T Kossmann, et al.
Pageof 8