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T Lashley

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Brain Pathology (Zurich, Switzerland)|April 15, 2006
Genetic alterations of the BRI2 gene: familial British and Danish dementiasJ Ghiso, A Rostagno, Y Tomidokoro, et al.
Neuropathology and Applied Neurobiology|February 20, 2008
Expression of BRI2 mRNA and protein in normal human brain and familial British dementia: its relevance to the pathogenesis of diseaseT Lashley, T Revesz, G Plant, et al.
Brain : a Journal of Neurology|April 19, 2002
Pathological, clinical and genetic heterogeneity in progressive supranuclear palsyH R Morris, G Gibb, R Katzenschlager, et al.
The Journal of Biological Chemistry|September 15, 2001
Systemic amyloid deposits in familial British dementiaJ A Ghiso, J Holton, L Miravalle, et al.
Neuropathology and Applied Neurobiology|September 16, 2006
Molecular chaperons, amyloid and preamyloid lesions in the BRI2 gene-related dementias: a morphological studyT Lashley, J L Holton, M M Verbeek, et al.
Amyloid : the International Journal of Experimental and Clinical Investigation : the Official Journal of the International Society of Amyloidosis|January 17, 2002
Chromosome 13 dementia syndromes as models of neurodegenerationJ Ghiso, T Révész, J Holton, et al.
Neuropathology and Applied Neurobiology|June 6, 2003
Pathological inclusion bodies in tauopathies contain distinct complements of tau with three or four microtubule-binding repeat domains as demonstrated by new specific monoclonal antibodiesR de Silva, T Lashley, G Gibb, et al.
The American Journal of Pathology|February 13, 2001
Regional distribution of amyloid-Bri deposition and its association with neurofibrillary degeneration in familial British dementiaJ L Holton, J Ghiso, T Lashley, et al.
Neuropathology and Applied Neurobiology|June 24, 2011
LRRK2 expression in idiopathic and G2019S positive Parkinson's disease subjects: a morphological and quantitative studyS Sharma, R Bandopadhyay, T Lashley, et al.
Annals of Neurology|November 18, 2000
Variant Alzheimer's disease with spastic paraparesis and cotton wool plaques is caused by PS-1 mutations that lead to exceptionally high amyloid-beta concentrationsH Houlden, M Baker, E McGowan, et al.
Pageof 2

Showing results (11-20 of 20) with videos related to

Sort By:
Pageof 2
You have reached the last page of results.This site can display upto 20 results.
Brain Pathology (Zurich, Switzerland)|April 15, 2006
Genetic alterations of the BRI2 gene: familial British and Danish dementiasJ Ghiso, A Rostagno, Y Tomidokoro, et al.
Neuropathology and Applied Neurobiology|February 20, 2008
Expression of BRI2 mRNA and protein in normal human brain and familial British dementia: its relevance to the pathogenesis of diseaseT Lashley, T Revesz, G Plant, et al.
Brain : a Journal of Neurology|April 19, 2002
Pathological, clinical and genetic heterogeneity in progressive supranuclear palsyH R Morris, G Gibb, R Katzenschlager, et al.
The Journal of Biological Chemistry|September 15, 2001
Systemic amyloid deposits in familial British dementiaJ A Ghiso, J Holton, L Miravalle, et al.
Neuropathology and Applied Neurobiology|September 16, 2006
Molecular chaperons, amyloid and preamyloid lesions in the BRI2 gene-related dementias: a morphological studyT Lashley, J L Holton, M M Verbeek, et al.
Amyloid : the International Journal of Experimental and Clinical Investigation : the Official Journal of the International Society of Amyloidosis|January 17, 2002
Chromosome 13 dementia syndromes as models of neurodegenerationJ Ghiso, T Révész, J Holton, et al.
Neuropathology and Applied Neurobiology|June 6, 2003
Pathological inclusion bodies in tauopathies contain distinct complements of tau with three or four microtubule-binding repeat domains as demonstrated by new specific monoclonal antibodiesR de Silva, T Lashley, G Gibb, et al.
The American Journal of Pathology|February 13, 2001
Regional distribution of amyloid-Bri deposition and its association with neurofibrillary degeneration in familial British dementiaJ L Holton, J Ghiso, T Lashley, et al.
Neuropathology and Applied Neurobiology|June 24, 2011
LRRK2 expression in idiopathic and G2019S positive Parkinson's disease subjects: a morphological and quantitative studyS Sharma, R Bandopadhyay, T Lashley, et al.
Annals of Neurology|November 18, 2000
Variant Alzheimer's disease with spastic paraparesis and cotton wool plaques is caused by PS-1 mutations that lead to exceptionally high amyloid-beta concentrationsH Houlden, M Baker, E McGowan, et al.
Pageof 2