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Blood|August 1, 1996
In vitro studies on cellular and molecular mechanisms of arsenic trioxide (As2O3) in the treatment of acute promyelocytic leukemia: As2O3 induces NB4 cell apoptosis with downregulation of Bcl-2 expression and modulation of PML-RAR alpha/PML proteinsG Q Chen, J Zhu, X G Shi, et al.Bone Marrow Transplantation|September 23, 2014
Delayed hematopoietic recovery after auto-SCT in patients receiving arsenic trioxide-based therapy for acute promyelocytic leukemia: a multi-center analysisG N Mannis, A C Logan, A D Leavitt, et al.Blood|May 1, 1997
Use of arsenic trioxide (As2O3) in the treatment of acute promyelocytic leukemia (APL): I. As2O3 exerts dose-dependent dual effects on APL cellsG Q Chen, X G Shi, W Tang, et al.Blood Cancer Journal|April 29, 2017
Frequent somatic mutations in epigenetic regulators in newly diagnosed chronic myeloid leukemiaE Togasaki, J Takeda, K Yoshida, et al.Leukemia|February 4, 2014
Comprehensive analysis of genetic alterations and their prognostic impacts in adult acute myeloid leukemia patientsR Kihara, Y Nagata, H Kiyoi, et al.Pageof 19