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Molecular and Cellular Biology|February 1, 1995
Interaction of Shc with Grb2 regulates association of Grb2 with mSOSK S Ravichandran, U Lorenz, S E Shoelson, et al.
Proceedings of the National Academy of Sciences of the United States of America|September 3, 1996
Lack of SHPTP1 results in src-family kinase hyperactivation and thymocyte hyperresponsivenessU Lorenz, K S Ravichandran, S J Burakoff, et al.
Molecular Cell|January 14, 1999
Cloning of p97/Gab2, the major SHP2-binding protein in hematopoietic cells, reveals a novel pathway for cytokine-induced gene activationH Gu, J C Pratt, S J Burakoff, et al.
The Journal of Biological Chemistry|November 1, 1996
CD43-specific activation of T cells induces association of CD43 to Fyn kinaseG Pedraza-Alva, L B Mérida, S J Burakoff, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 1, 1976
Inhibition of cell-mediated cytolysis of trinitrophenyl-derivatized target cells by alloantisera directed to the products of the K and D loci of the H-2 complexS J Burakoff, R N Germain, M E Dorf, et al.
Journal of Immunology (Baltimore, Md. : 1950)|May 1, 1984
LFA-1, LFA-2, and LFA-3 antigens are involved in CTL-target conjugationA M Krensky, E Robbins, T A Springer, et al.
The Journal of Experimental Medicine|June 1, 1983
Analysis of cross-reactive antigen-specific T cell clones. Specific recognition of two major histocompatibility complex (MHC) and two non-MHC antigens by a single cloneA Ben-Nun, Z Lando, M E Dorf, et al.
Journal of Immunology (Baltimore, Md. : 1950)|December 1, 1981
Ir gene control of the cytotoxic T lymphocyte response to Sendai virus: H-2k mice are low responders to SendaiM E Shapiro, S J Burakoff, B Benacerraf, et al.
Infection and Immunity|February 1, 1981
Type-specific reovirus antiserum blocks the cytotoxic T-cell-target cell interaction: evidence for the association of the viral hemagglutinin of a nonenveloped virus with the cell surfaceR Finberg, H L Weiner, S J Burakoff, et al.
Cancer|February 1, 1992
Lymphoid irradiation results in long-term increases in natural killer cells in patients treated for Hodgkin's diseaseR M Macklis, P M Mauch, S J Burakoff, et al.
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