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T R Golub

Showing results (1-10 of 35) with videos related to

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Hematology/Oncology Clinics of North America|January 27, 1998
TEL gene rearrangements in myeloid malignancyT R Golub
The New England Journal of Medicine|February 24, 2001
Genome-wide views of cancerT R Golub
Current Opinion in Hematology|September 19, 2001
Genomic approaches to the pathogenesis of hematologic malignancyT R Golub
Cell|April 22, 1994
Fusion of PDGF receptor beta to a novel ets-like gene, tel, in chronic myelomonocytic leukemia with t(5;12) chromosomal translocationT R Golub, G F Barker, M Lovett, et al.
Current Topics in Microbiology and Immunology|January 1, 1996
Involvement of the TEL gene in hematologic malignancy by diverse molecular genetic mechanismsT R Golub, G F Barker, K Stegmaier, et al.
Current Topics in Microbiology and Immunology|January 1, 1997
The TEL gene contributes to the pathogenesis of myeloid and lymphoid leukemias by diverse molecular genetic mechanismsT R Golub, G F Barker, K Stegmaier, et al.
Oncogene|September 19, 1996
p210BCR/ABL, p190BCR/ABL, and TEL/ABL activate similar signal transduction pathways in hematopoietic cell linesK Okuda, T R Golub, D G Gilliland, et al.
Nature|August 22, 2000
Genomic analysis of metastasis reveals an essential role for RhoCE A Clark, T R Golub, E S Lander, et al.
Proceedings of the National Academy of Sciences of the United States of America|October 12, 2000
Discovering functional relationships between RNA expression and chemotherapeutic susceptibility using relevance networksA J Butte, P Tamayo, D Slonim, et al.
Proceedings of the National Academy of Sciences of the United States of America|December 10, 1996
The TEL/platelet-derived growth factor beta receptor (PDGF beta R) fusion in chronic myelomonocytic leukemia is a transforming protein that self-associates and activates PDGF beta R kinase-dependent signaling pathwaysM Carroll, M H Tomasson, G F Barker, et al.
Pageof 4

Showing results (1-10 of 35) with videos related to

Sort By:
Pageof 4
Hematology/Oncology Clinics of North America|January 27, 1998
TEL gene rearrangements in myeloid malignancyT R Golub
The New England Journal of Medicine|February 24, 2001
Genome-wide views of cancerT R Golub
Current Opinion in Hematology|September 19, 2001
Genomic approaches to the pathogenesis of hematologic malignancyT R Golub
Cell|April 22, 1994
Fusion of PDGF receptor beta to a novel ets-like gene, tel, in chronic myelomonocytic leukemia with t(5;12) chromosomal translocationT R Golub, G F Barker, M Lovett, et al.
Current Topics in Microbiology and Immunology|January 1, 1996
Involvement of the TEL gene in hematologic malignancy by diverse molecular genetic mechanismsT R Golub, G F Barker, K Stegmaier, et al.
Current Topics in Microbiology and Immunology|January 1, 1997
The TEL gene contributes to the pathogenesis of myeloid and lymphoid leukemias by diverse molecular genetic mechanismsT R Golub, G F Barker, K Stegmaier, et al.
Oncogene|September 19, 1996
p210BCR/ABL, p190BCR/ABL, and TEL/ABL activate similar signal transduction pathways in hematopoietic cell linesK Okuda, T R Golub, D G Gilliland, et al.
Nature|August 22, 2000
Genomic analysis of metastasis reveals an essential role for RhoCE A Clark, T R Golub, E S Lander, et al.
Proceedings of the National Academy of Sciences of the United States of America|October 12, 2000
Discovering functional relationships between RNA expression and chemotherapeutic susceptibility using relevance networksA J Butte, P Tamayo, D Slonim, et al.
Proceedings of the National Academy of Sciences of the United States of America|December 10, 1996
The TEL/platelet-derived growth factor beta receptor (PDGF beta R) fusion in chronic myelomonocytic leukemia is a transforming protein that self-associates and activates PDGF beta R kinase-dependent signaling pathwaysM Carroll, M H Tomasson, G F Barker, et al.
Pageof 4