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Journal of Neurochemistry|March 11, 2004
Mitochondrial calcium, oxidative stress and apoptosis in a neurodegenerative disease model induced by 3-nitropropionic acidT R Rosenstock, A C P Carvalho, A Jurkiewicz, et al.Pharmacology, Biochemistry, and Behavior|September 4, 2008
Old mice present increased levels of succinate dehydrogenase activity and lower vulnerability to dyskinetic effects of 3-nitropropionic acidT R Rosenstock, V C Abílio, R Frussa-Filho, et al.The European Journal of Neuroscience|July 9, 2010
Glutamate-induced alterations in Ca2+ signaling are modulated by mitochondrial Ca2+ handling capacity in brain slices of R6/1 transgenic miceT R Rosenstock, C R A Bertoncini, A V Teles, et al.Cell Death and Differentiation|October 23, 2004
Bax affects intracellular Ca2+ stores and induces Ca2+ wave propagationA C P Carvalho, J Sharpe, T R Rosenstock, et al.Brazilian Journal of Medical and Biological Research = Revista Brasileira De Pesquisas Medicas E Biologicas|February 4, 2003
Mitochondria, calcium and pro-apoptotic proteins as mediators in cell death signalingS S Smaili, Y-T Hsu, A C P Carvalho, et al.Neuroscience Letters|May 13, 2008
Increase in bax expression and apoptosis are associated in Huntington's disease progressionA V F F Teles, T R Rosenstock, C S Okuno, et al.Pageof 1