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The American Journal of Physiology|October 1, 1975
Metabolic background for glucose tolerance: mechanism for epinephrine-induced impairmentH Skikama, M UiThe Journal of Biological Chemistry|January 25, 1984
[3H]GDP release from rat and hamster adipocyte membranes independently linked to receptors involved in activation or inhibition of adenylate cyclase. Differential susceptibility to two bacterial toxinsT Murayama, M UiThe Journal of Biological Chemistry|March 10, 1983
Loss of the inhibitory function of the guanine nucleotide regulatory component of adenylate cyclase due to its ADP ribosylation by islet-activating protein, pertussis toxin, in adipocyte membranesT Murayama, M UiArchives of Biochemistry and Biophysics|May 1, 1984
Predominance of beta-adrenergic over alpha-adrenergic receptor functions involved in phosphorylase activation in liver cells of cholestatic ratsF Okajima, M UiThe Journal of Biological Chemistry|November 25, 1984
ADP-ribosylation of the specific membrane protein by islet-activating protein, pertussis toxin, associated with inhibition of a chemotactic peptide-induced arachidonate release in neutrophils. A possible role of the toxin substrate in Ca2+-mobilizing biosignalingF Okajima, M UiThe Journal of Biological Chemistry|October 25, 1980
Slow interaction of islet-activating protein with pancreatic islets during primary culture to cause reversal of alpha-adrenergic inhibition of insulin secretionT Katada, M UiJapanese Journal of Pharmacology|March 1, 1984
Effects of somatostatin on liver glycogen and fat metabolism in vivoM Ogihara, M UiBiochemical Pharmacology|November 15, 1983
Suppression of passive cutaneous anaphylaxis by pertussis toxin, an islet-activating protein, as a result of inhibition of histamine release from mast cellsT Nakamura, M UiEndocrinology|October 1, 1977
Perfusion of the pancreas isolated from pertussis-sensitized rats: potentiation of insulin secretory responses due to beta-adrenergic stimulationT Katada, M UiThe Biochemical Journal|August 15, 1979
Metabolism of glucose in hyper- and hypo-thyroid rats in vivo. Glucose-turnover values and futile-cycle activities obtained with 14C- and 3H-labelled glucoseF Okajima, M UiPageof 44