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T W Loo

Showing results (51-60 of 63) with videos related to

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Cell Motility and the Cytoskeleton|January 1, 1989
Expression and mutation of Ca2+ ATPases of the sarcoplasmic reticulumK Maruyama, D M Clarke, J Fujii, et al.
Biochimica Et Biophysica Acta|September 7, 1983
Interaction of Escherichia coli F1-ATPase with dicyclohexylcarbodiimide-binding polypeptideT W Loo, H Stan-Lotter, D MacKenzie, et al.
Journal of Virological Methods|May 1, 1986
Detection of antibodies to individual proteins of rubella virusT W Loo, I MacDonald, D M Clarke, et al.
The Journal of Biological Chemistry|July 5, 1989
Functional consequences of glutamate, aspartate, glutamine, and asparagine mutations in the stalk sector of the Ca2+-ATPase of sarcoplasmic reticulumD M Clarke, K Maruyama, T W Loo, et al.
The Journal of Physiology|August 1, 1995
Failure of P-glycoprotein (MDR1) expressed in Xenopus oocytes to produce swelling-activated chloride channel activityX K Morin, T D Bond, T W Loo, et al.
The Journal of Biological Chemistry|August 5, 1989
Functional consequences of alterations to amino acids located in the catalytic center (isoleucine 348 to threonine 357) and nucleotide-binding domain of the Ca2+-ATPase of sarcoplasmic reticulumK Maruyama, D M Clarke, J Fujii, et al.
The Journal of Biological Chemistry|November 1, 1996
Cytoplasmic loop three of cystic fibrosis transmembrane conductance regulator contributes to regulation of chloride channel activityF S Seibert, P Linsdell, T W Loo, et al.
The Journal of Biological Chemistry|August 25, 1993
Functional consequences of alterations to hydrophobic amino acids located in the M4 transmembrane sector of the Ca(2+)-ATPase of sarcoplasmic reticulumD M Clarke, T W Loo, W J Rice, et al.
The Journal of Biological Chemistry|June 21, 1996
Disease-associated mutations in the fourth cytoplasmic loop of cystic fibrosis transmembrane conductance regulator compromise biosynthetic processing and chloride channel activityF S Seibert, P Linsdell, T W Loo, et al.
Biochemistry|October 8, 1997
Disease-associated mutations in cytoplasmic loops 1 and 2 of cystic fibrosis transmembrane conductance regulator impede processing or opening of the channelF S Seibert, Y Jia, C J Mathews, et al.
Pageof 7

Showing results (51-60 of 63) with videos related to

Sort By:
Pageof 7
Cell Motility and the Cytoskeleton|January 1, 1989
Expression and mutation of Ca2+ ATPases of the sarcoplasmic reticulumK Maruyama, D M Clarke, J Fujii, et al.
Biochimica Et Biophysica Acta|September 7, 1983
Interaction of Escherichia coli F1-ATPase with dicyclohexylcarbodiimide-binding polypeptideT W Loo, H Stan-Lotter, D MacKenzie, et al.
Journal of Virological Methods|May 1, 1986
Detection of antibodies to individual proteins of rubella virusT W Loo, I MacDonald, D M Clarke, et al.
The Journal of Biological Chemistry|July 5, 1989
Functional consequences of glutamate, aspartate, glutamine, and asparagine mutations in the stalk sector of the Ca2+-ATPase of sarcoplasmic reticulumD M Clarke, K Maruyama, T W Loo, et al.
The Journal of Physiology|August 1, 1995
Failure of P-glycoprotein (MDR1) expressed in Xenopus oocytes to produce swelling-activated chloride channel activityX K Morin, T D Bond, T W Loo, et al.
The Journal of Biological Chemistry|August 5, 1989
Functional consequences of alterations to amino acids located in the catalytic center (isoleucine 348 to threonine 357) and nucleotide-binding domain of the Ca2+-ATPase of sarcoplasmic reticulumK Maruyama, D M Clarke, J Fujii, et al.
The Journal of Biological Chemistry|November 1, 1996
Cytoplasmic loop three of cystic fibrosis transmembrane conductance regulator contributes to regulation of chloride channel activityF S Seibert, P Linsdell, T W Loo, et al.
The Journal of Biological Chemistry|August 25, 1993
Functional consequences of alterations to hydrophobic amino acids located in the M4 transmembrane sector of the Ca(2+)-ATPase of sarcoplasmic reticulumD M Clarke, T W Loo, W J Rice, et al.
The Journal of Biological Chemistry|June 21, 1996
Disease-associated mutations in the fourth cytoplasmic loop of cystic fibrosis transmembrane conductance regulator compromise biosynthetic processing and chloride channel activityF S Seibert, P Linsdell, T W Loo, et al.
Biochemistry|October 8, 1997
Disease-associated mutations in cytoplasmic loops 1 and 2 of cystic fibrosis transmembrane conductance regulator impede processing or opening of the channelF S Seibert, Y Jia, C J Mathews, et al.
Pageof 7