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Genomics|April 5, 2003
Bilateral congenital cataracts result from a gain-of-function mutation in the gene for aquaporin-0 in miceTadashi Okamura, Ichiro Miyoshi, Kazuhiro Takahashi, et al.
Biochemical and Biophysical Research Communications|April 11, 2021
A single amino acid substitution in PRKDC is a determinant of sensitivity to Adriamycin-induced renal injury in mouseMasaki Watanabe, Yuki Takahashi, Koki Hiura, et al.
Journal of Chromatography. B, Analytical Technologies in the Biomedical and Life Sciences|September 21, 2010
Simultaneous two-dimensional HPLC determination of free D-serine and D-alanine in the brain and periphery of mutant rats lacking D-amino-acid oxidaseYurika Miyoshi, Kenji Hamase, Tadashi Okamura, et al.
The Journal of Veterinary Medical Science|June 2, 2016
Functional validation of tensin2 SH2-PTB domain by CRISPR/Cas9-mediated genome editingKiyoma Marusugi, Kenta Nakano, Hayato Sasaki, et al.
Journal of Diabetes Investigation|July 11, 2018
Novel murine model of congenital diabetes: The insulin hyposecretion mouseKenta Nakano, Rieko Yanobu-Takanashi, Yuki Takahashi, et al.
Antiviral Research|December 10, 2009
TACE antagonists blocking ACE2 shedding caused by the spike protein of SARS-CoV are candidate antiviral compoundsShiori Haga, Noriyo Nagata, Tadashi Okamura, et al.
International Heart Journal|May 13, 2016
Oxidative Stress Induced Ventricular Arrhythmia and Impairment of Cardiac Function in Nos1ap Deleted MiceKoji Sugiyama, Tetsuo Sasano, Junko Kurokawa, et al.
International Immunology|July 18, 2022
A protein kinase D inhibitor suppresses AKT on T cells and antagonizes cancer immunotherapy by anti-PD-1Kazuhide Miyamoto, Hodaka Hayabuchi, Yukiko Tokifuji, et al.
Journal of Pharmaceutical and Biomedical Analysis|February 8, 2026
Three-dimensional high-performance liquid chromatographic determination of serine, threonine and allothreonine enantiomers in the d-amino acid oxidase deficient mice and ratsMai Oyaide, Takeyuki Akita, Chiharu Ishii, et al.
Experimental Animals|November 15, 2019
Deletion of the Tensin2 SH2-PTB domain, but not the loss of its PTPase activity, induces podocyte injury in FVB/N mouse strainHayato Sasaki, Yuki Takahashi, Tsubasa Ogawa, et al.
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