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Taiichi Katayama

Showing results (11-20 of 93) with videos related to

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The European Journal of Neuroscience|August 20, 2003
GRP94 (94 kDa glucose-regulated protein) suppresses ischemic neuronal cell death against ischemia/reperfusion injuryYoshio Bando, Taiichi Katayama, Kousuke Kasai, et al.
Neuroscience Letters|March 24, 2004
Apoptosis induced by endoplasmic reticulum stress depends on activation of caspase-3 via caspase-12Junichi Hitomi, Taiichi Katayama, Manabu Taniguchi, et al.
Biochemical and Biophysical Research Communications|November 5, 2022
TMEM67 is required for the gating function of the transition zone that controls entry of membrane-associated proteins ARL13B and INPP5E into primary ciliaZhuoma Yinsheng, Ko Miyoshi, Yuanyuan Qin, et al.
Molecular Neurobiology|May 14, 2022
SUMO1 Modification of Tau in Progressive Supranuclear PalsyHironori Takamura, Yoshiaki Nakayama, Hidefumi Ito, et al.
Frontiers in Psychiatry|April 10, 2026
Animal models for the study of ADHD: the need for next-generation modelsHiroki Furuie, Taichi Hatakeyama, Wakana Harigai, et al.
Neuroscience Letters|July 23, 2002
The cytosolic inclusion bodies that consist of splice variants that lack exon 5 of the presenilin-2 gene differ obviously from Hirano bodies observed in the brain from sporadic cases of Alzheimer's disease patientsTakayuki Manabe, Taiichi Katayama, Naoya Sato, et al.
Neurochemistry International|October 19, 2007
Presenilin-1 mutation activates the signaling pathway of caspase-4 in endoplasmic reticulum stress-induced apoptosisFutoshi Yukioka, Shinsuke Matsuzaki, Keisuke Kawamoto, et al.
Biochemical and Biophysical Research Communications|May 15, 2007
Monomeric A beta and metals reduce their cytotoxicities to each otherShinsuke Matsuzaki, Yuichi Yasuda, Shinya Kobayashi, et al.
Neurochemistry International|September 7, 2010
Familial amyotrophic lateral sclerosis (FALS)-linked SOD1 mutation accelerates neuronal cell death by activating cleavage of caspase-4 under ER stress in an in vitro model of FALSYoshihisa Koyama, Toru Hiratsuka, Shinsuke Matsuzaki, et al.
Neurobiology of Disease|January 15, 2005
RA410/Sly1 suppresses MPP+ and 6-hydroxydopamine-induced cell death in SH-SY5Y cellsYoshio Bando, Taiichi Katayama, Manabu Taniguchi, et al.
Pageof 10

Showing results (11-20 of 93) with videos related to

Sort By:
Pageof 10
The European Journal of Neuroscience|August 20, 2003
GRP94 (94 kDa glucose-regulated protein) suppresses ischemic neuronal cell death against ischemia/reperfusion injuryYoshio Bando, Taiichi Katayama, Kousuke Kasai, et al.
Neuroscience Letters|March 24, 2004
Apoptosis induced by endoplasmic reticulum stress depends on activation of caspase-3 via caspase-12Junichi Hitomi, Taiichi Katayama, Manabu Taniguchi, et al.
Biochemical and Biophysical Research Communications|November 5, 2022
TMEM67 is required for the gating function of the transition zone that controls entry of membrane-associated proteins ARL13B and INPP5E into primary ciliaZhuoma Yinsheng, Ko Miyoshi, Yuanyuan Qin, et al.
Molecular Neurobiology|May 14, 2022
SUMO1 Modification of Tau in Progressive Supranuclear PalsyHironori Takamura, Yoshiaki Nakayama, Hidefumi Ito, et al.
Frontiers in Psychiatry|April 10, 2026
Animal models for the study of ADHD: the need for next-generation modelsHiroki Furuie, Taichi Hatakeyama, Wakana Harigai, et al.
Neuroscience Letters|July 23, 2002
The cytosolic inclusion bodies that consist of splice variants that lack exon 5 of the presenilin-2 gene differ obviously from Hirano bodies observed in the brain from sporadic cases of Alzheimer's disease patientsTakayuki Manabe, Taiichi Katayama, Naoya Sato, et al.
Neurochemistry International|October 19, 2007
Presenilin-1 mutation activates the signaling pathway of caspase-4 in endoplasmic reticulum stress-induced apoptosisFutoshi Yukioka, Shinsuke Matsuzaki, Keisuke Kawamoto, et al.
Biochemical and Biophysical Research Communications|May 15, 2007
Monomeric A beta and metals reduce their cytotoxicities to each otherShinsuke Matsuzaki, Yuichi Yasuda, Shinya Kobayashi, et al.
Neurochemistry International|September 7, 2010
Familial amyotrophic lateral sclerosis (FALS)-linked SOD1 mutation accelerates neuronal cell death by activating cleavage of caspase-4 under ER stress in an in vitro model of FALSYoshihisa Koyama, Toru Hiratsuka, Shinsuke Matsuzaki, et al.
Neurobiology of Disease|January 15, 2005
RA410/Sly1 suppresses MPP+ and 6-hydroxydopamine-induced cell death in SH-SY5Y cellsYoshio Bando, Taiichi Katayama, Manabu Taniguchi, et al.
Pageof 10