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Cold Spring Harbor Perspectives in Medicine|June 8, 2012
Proteolytic degradation of amyloid β-proteinTakaomi Saido, Malcolm A LeissringThe Journal of Comparative Neurology|August 10, 2004
Calpain activity in the amikacin-damaged rat cochleaSabine Ladrech, Matthieu Guitton, Takaomi Saido, et al.Seminars in Cell & Developmental Biology|March 16, 2022
Presubiculum principal cells are preserved from degeneration in knock-in APP/TAU mouse models of Alzheimer's diseaseAnam Islam, Takashi Saito, Takaomi Saido, et al.Journal of Alzheimer'S Disease : JAD|July 5, 2021
Neuronal Cell Cycle Re-Entry Enhances Neuropathological Features in AppNLF Knock-In MiceTomás Barrett, Katherine A Stangis, Takashi Saito, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|October 2, 2015
Neuronal Store-Operated Calcium Entry and Mushroom Spine Loss in Amyloid Precursor Protein Knock-In Mouse Model of Alzheimer's DiseaseHua Zhang, Lili Wu, Ekaterina Pchitskaya, et al.Scientific Reports|February 3, 2022
Astrocytes deficient in circadian clock gene Bmal1 show enhanced activation responses to amyloid-beta pathology without changing plaque burdenCelia A McKee, Jiyeon Lee, Yuqi Cai, et al.Fluids and Barriers of the CNS|September 13, 2023
Proteomic alterations in the brain and blood-brain barrier during brain Aβ accumulation in an APP knock-in mouse model of Alzheimer's diseaseShingo Ito, Ryotaro Yagi, Seiryo Ogata, et al.Alzheimer'S Research & Therapy|August 26, 2020
The two faces of synaptic failure in App<sup>NL-G-F</sup> knock-in miceAmira Latif-Hernandez, Victor Sabanov, Tariq Ahmed, et al.Elife|March 25, 2025
Aβ-driven nuclear pore complex dysfunction alters activation of necroptosis proteins in a mouse model of Alzheimer's diseaseVibhavari Aysha Bansal, Jia Min Tan, Hui Rong Soon, et al.Pharmacological Research|September 11, 2002
In vivo glioma growth requires host-derived matrix metalloproteinase 2 for maintenance of angioarchitectureMegumi Takahashi, Shinjiro Fukami, Nobuhisa Iwata, et al.Pageof 5