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Biomedical Research (Tokyo, Japan)|May 10, 2011
Pancreatic β-cells are generated by neogenesis from non-β-cells after birthKorefumi Nakamura, Kohtaro Minami, Kanako Tamura, et al.Clinical Case Reports|July 11, 2018
Anomalous aortic origin of the right coronary artery with functional ischemia determined with fractional flow reserve derived from computed tomographyTakashi Miki, Toru Miyoshi, Atsuyuki Watanabe, et al.Experimental Biology and Medicine (Maywood, N.J.)|November 12, 2003
Ca2+ influx does not trigger glucose-induced traffic of the insulin granules and alteration of their distributionIchiro Niki, Tae Niwa, Wei Yu, et al.Endocrinology|September 13, 2008
Glucose controls cytosolic Ca2+ and insulin secretion in mouse islets lacking adenosine triphosphate-sensitive K+ channels owing to a knockout of the pore-forming subunit Kir6.2Magalie A Ravier, Myriam Nenquin, Takashi Miki, et al.Diabetes|June 29, 2006
Spontaneous recovery from hyperglycemia by regeneration of pancreatic beta-cells in Kir6.2G132S transgenic miceKazunobu Oyama, Kohtaro Minami, Katsuhiko Ishizaki, et al.Journal of Pharmacological Sciences|September 21, 2017
Electrophysiological analyses of transgenic mice overexpressing KCNJ8 with S422L mutation in cardiomyocytesYasuhiro Watanabe, Akio Matsumoto, Takashi Miki, et al.Iscience|September 23, 2024
Mosaic ablation of pancreatic β cells induces de-differentiation and repetitive proliferation of residual β cells in adult miceRyo Hatano, Xilin Zhang, Eunyoung Lee, et al.The European Journal of Neuroscience|April 21, 2007
Disruption of Kir6.2-containing ATP-sensitive potassium channels impairs maintenance of hypoxic gasping in miceAkari Miyake, Katsuya Yamada, Tomohiro Kosaka, et al.Climacteric : the Journal of the International Menopause Society|June 12, 2024
Phase II study of fezolinetant for treatment of vasomotor symptoms associated with menopause in JapanKiyoshi Takamatsu, Takashi Miki, Kentaro Miyazaki, et al.Nature Neuroscience|November 22, 2005
K-ATP channels promote the differential degeneration of dopaminergic midbrain neuronsBirgit Liss, Olga Haeckel, Johannes Wildmann, et al.Pageof 21