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Journal of the American College of Cardiology|October 24, 2002
Infarct size limitation by nicorandil: roles of mitochondrial K(ATP) channels, sarcolemmal K(ATP) channels, and protein kinase CAkihito Tsuchida, Tetsuji Miura, Masaya Tanno, et al.
American Journal of Physiology. Heart and Circulatory Physiology|April 17, 2012
Role of connexin-43 in protective PI3K-Akt-GSK-3β signaling in cardiomyocytesSatoko Ishikawa, Atsushi Kuno, Masaya Tanno, et al.
Medicine|June 23, 2021
Characteristics of patients with emergency attendance for severe hypoglycemia and hyperglycemia in a general hospital in JapanKeisuke Endo, Takahito Itoh, Masaya Tanno, et al.
Open Heart|January 27, 2017
Distinct risk factors of atrial fibrillation in patients with and without coronary artery disease: a cross-sectional analysis of the BOREAS-CAG Registry dataNaoto Murakami, Masaya Tanno, Nobuaki Kokubu, et al.
Hypertension (Dallas, Tex. : 1979)|November 10, 2010
Hypertensive hypertrophied myocardium is vulnerable to infarction and refractory to erythropoietin-induced protectionToshiyuki Yano, Takayuki Miki, Masaya Tanno, et al.
Diabetes|September 17, 2009
Endoplasmic reticulum stress in diabetic hearts abolishes erythropoietin-induced myocardial protection by impairment of phospho-glycogen synthase kinase-3beta-mediated suppression of mitochondrial permeability transitionTakayuki Miki, Tetsuji Miura, Hiroyuki Hotta, et al.
Journal of Molecular and Cellular Cardiology|October 13, 2007
Modulation of the mitochondrial permeability transition pore complex in GSK-3beta-mediated myocardial protectionMasahiro Nishihara, Tetsuji Miura, Takayuki Miki, et al.
Physiological Reports|June 23, 2018
Empagliflozin normalizes the size and number of mitochondria and prevents reduction in mitochondrial size after myocardial infarction in diabetic heartsMasashi Mizuno, Atsushi Kuno, Toshiyuki Yano, et al.
Cardiovascular Pathology : the Official Journal of the Society for Cardiovascular Pathology|September 20, 2005
Intracardiac fibroblasts, but not bone marrow derived cells, are the origin of myofibroblasts in myocardial infarct repairToshiyuki Yano, Tetsuji Miura, Yoshihiro Ikeda, et al.
Internal Medicine (Tokyo, Japan)|September 26, 2017
The Serum Level of KL-6 Is Associated with the Risk of Insulin Resistance and New-onset Diabetes Mellitus: The Tanno-Sobetsu StudyHiroshi Akasaka, Hirofumi Ohnishi, Yoshifumi Narita, et al.
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