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Proceedings of the National Academy of Sciences of the United States of America|February 11, 2014
Transmitochondrial mice as models for primary prevention of diseases caused by mutation in the tRNA(Lys) geneAkinori Shimizu, Takayuki Mito, Chisato Hayashi, et al.Plos One|February 19, 2013
Mitochondrial DNA mutations in mutator mice confer respiration defects and B-cell lymphoma developmentTakayuki Mito, Yoshiaki Kikkawa, Akinori Shimizu, et al.Biochemical and Biophysical Research Communications|December 17, 2014
Transmitochondrial mito-miceΔ and mtDNA mutator mice, but not aged mice, share the same spectrum of musculoskeletal disordersTakayuki Mito, Hikari Ishizaki, Michiko Suzuki, et al.Scientific Reports|January 12, 2018
Mice deficient in the Shmt2 gene have mitochondrial respiration defects and are embryonic lethalHaruna Tani, Sakiko Ohnishi, Hiroshi Shitara, et al.Scientific Reports|May 23, 2015
Epigenetic regulation of the nuclear-coded GCAT and SHMT2 genes confers human age-associated mitochondrial respiration defectsOsamu Hashizume, Sakiko Ohnishi, Takayuki Mito, et al.Cell Reports|May 17, 2025
De novo serine biosynthesis is protective in mitochondrial diseaseChristopher B Jackson, Anastasiia Marmyleva, Geoffray Monteuuis, et al.Nature|April 3, 2024
Ancestral allele of DNA polymerase gamma modifies antiviral toleranceYilin Kang, Jussi Hepojoki, Rocio Sartori Maldonado, et al.Pageof 2