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Annals of the Rheumatic Diseases|May 31, 2015
RANKL expressed on synovial fibroblasts is primarily responsible for bone erosions during joint inflammationLynett Danks, Noriko Komatsu, Matteo M Guerrini, et al.Proceedings of the National Academy of Sciences of the United States of America|February 6, 2010
Blimp1-mediated repression of negative regulators is required for osteoclast differentiationKeizo Nishikawa, Tomoki Nakashima, Mikihito Hayashi, et al.Modern Rheumatology|February 6, 2007
Scientific basis for the efficacy of combined use of antirheumatic drugs against bone destruction in rheumatoid arthritisAyako Suematsu, Yasuhito Tajiri, Tomoki Nakashima, et al.Immunity|December 19, 2015
Inhibition of the TNF Family Cytokine RANKL Prevents Autoimmune Inflammation in the Central Nervous SystemMatteo M Guerrini, Kazuo Okamoto, Noriko Komatsu, et al.Nature Medicine|March 8, 2005
The molecular scaffold Gab2 is a crucial component of RANK signaling and osteoclastogenesisTeiji Wada, Tomoki Nakashima, Antonio J Oliveira-dos-Santos, et al.Biochemical and Biophysical Research Communications|June 22, 2011
The role of the BH3-only protein Noxa in bone homeostasisErik Idrus, Tomoki Nakashima, Ling Wang, et al.The Journal of Biological Chemistry|September 19, 2012
Intracellular and extracellular ATP coordinately regulate the inverse correlation between osteoclast survival and bone resorptionTsuyoshi Miyazaki, Mitsuyasu Iwasawa, Tomoki Nakashima, et al.Physiological Reviews|August 18, 2017
Osteoimmunology: The Conceptual Framework Unifying the Immune and Skeletal SystemsKazuo Okamoto, Tomoki Nakashima, Masahiro Shinohara, et al.Nature Immunology|October 17, 2018
Arginine methylation controls the strength of γc-family cytokine signaling in T cell maintenanceMaia Inoue, Kazuo Okamoto, Asuka Terashima, et al.Nature Medicine|December 24, 2013
Pathogenic conversion of Foxp3+ T cells into TH17 cells in autoimmune arthritisNoriko Komatsu, Kazuo Okamoto, Shinichiro Sawa, et al.Pageof 9