Search research articles
Contact Us
Filters
Showing results (21-30 of 91) with videos related to
Page
of 10
Sort By:
Cell Cycle (Georgetown, Tex.)
|
August 23, 2008
RecQ family helicases in genome stability: lessons from gene disruption studies in DT40 cells
Masayuki Seki, Makoto Otsuki, Yutaka Ishii, et al.
Molecular and Cellular Biology
|
March 17, 2010
RecQL5 promotes genome stabilization through two parallel mechanisms--interacting with RNA polymerase II and acting as a helicase
M Nurul Islam, David Fox, Rong Guo, et al.
Biochemical and Biophysical Research Communications
|
January 16, 2007
Chl1 and Ctf4 are required for damage-induced recombinations
Hideaki Ogiwara, Ayako Ui, Mong Sing Lai, et al.
Journal of Cell Science
|
July 16, 2002
Focus-formation of replication protein A, activation of checkpoint system and DNA repair synthesis induced by DNA double-strand breaks in Xenopus egg extract
Takayuki Kobayashi, Shusuke Tada, Takashi Tsuyama, et al.
Biochemical and Biophysical Research Communications
|
June 21, 2011
The role of SNM1 family nucleases in etoposide-induced apoptosis
Yoshifumi Hosono, Takuya Abe, Masamichi Ishiai, et al.
DNA Repair
|
January 28, 2003
Budding yeast mms4 is epistatic with rad52 and the function of Mms4 can be replaced by a bacterial Holliday junction resolvase
Nao Odagiri, Masayuki Seki, Fumitoshi Onoda, et al.
Nucleic Acids Research
|
July 15, 2006
Dpb11, the budding yeast homolog of TopBP1, functions with the checkpoint clamp in recombination repair
Hideaki Ogiwara, Ayako Ui, Fumitoshi Onoda, et al.
Nucleic Acids Research
|
July 20, 2007
Ctf18 is required for homologous recombination-mediated double-strand break repair
Hideaki Ogiwara, Takashi Ohuchi, Ayako Ui, et al.
Biological & Pharmaceutical Bulletin
|
May 8, 2019
WRNIP1 Controls the Amount of PrimPol
Akari Yoshimura, Mizuho Oikawa, Hitomi Jinbo, et al.
Biochimica Et Biophysica Acta
|
January 25, 2011
The N-terminal region of RECQL4 lacking the helicase domain is both essential and sufficient for the viability of vertebrate cells. Role of the N-terminal region of RECQL4 in cells
Takuya Abe, Akari Yoshimura, Yoshifumi Hosono, et al.
Page
of 10
Search research articles
Search
Showing results (21-30 of 91) with videos related to
Sort By:
Page
of 10
Cell Cycle (Georgetown, Tex.)
|
August 23, 2008
RecQ family helicases in genome stability: lessons from gene disruption studies in DT40 cells
Masayuki Seki, Makoto Otsuki, Yutaka Ishii, et al.
Molecular and Cellular Biology
|
March 17, 2010
RecQL5 promotes genome stabilization through two parallel mechanisms--interacting with RNA polymerase II and acting as a helicase
M Nurul Islam, David Fox, Rong Guo, et al.
Biochemical and Biophysical Research Communications
|
January 16, 2007
Chl1 and Ctf4 are required for damage-induced recombinations
Hideaki Ogiwara, Ayako Ui, Mong Sing Lai, et al.
Journal of Cell Science
|
July 16, 2002
Focus-formation of replication protein A, activation of checkpoint system and DNA repair synthesis induced by DNA double-strand breaks in Xenopus egg extract
Takayuki Kobayashi, Shusuke Tada, Takashi Tsuyama, et al.
Biochemical and Biophysical Research Communications
|
June 21, 2011
The role of SNM1 family nucleases in etoposide-induced apoptosis
Yoshifumi Hosono, Takuya Abe, Masamichi Ishiai, et al.
DNA Repair
|
January 28, 2003
Budding yeast mms4 is epistatic with rad52 and the function of Mms4 can be replaced by a bacterial Holliday junction resolvase
Nao Odagiri, Masayuki Seki, Fumitoshi Onoda, et al.
Nucleic Acids Research
|
July 15, 2006
Dpb11, the budding yeast homolog of TopBP1, functions with the checkpoint clamp in recombination repair
Hideaki Ogiwara, Ayako Ui, Fumitoshi Onoda, et al.
Nucleic Acids Research
|
July 20, 2007
Ctf18 is required for homologous recombination-mediated double-strand break repair
Hideaki Ogiwara, Takashi Ohuchi, Ayako Ui, et al.
Biological & Pharmaceutical Bulletin
|
May 8, 2019
WRNIP1 Controls the Amount of PrimPol
Akari Yoshimura, Mizuho Oikawa, Hitomi Jinbo, et al.
Biochimica Et Biophysica Acta
|
January 25, 2011
The N-terminal region of RECQL4 lacking the helicase domain is both essential and sufficient for the viability of vertebrate cells. Role of the N-terminal region of RECQL4 in cells
Takuya Abe, Akari Yoshimura, Yoshifumi Hosono, et al.
Page
of 10