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Journal of Cell Science|November 24, 2019
The balance of forces generated by kinesins controls spindle polarity and chromosomal heterogeneity in tetraploid cellsSei Shu, Makoto Iimori, Takeshi Wakasa, et al.Cancer Science|January 27, 2026
IAP Antagonists Selectively Eliminate Therapy-Induced Senescent Cancer Cells via TNFα-Independent ApoptosisHiroaki Ochiiwa, Takeshi Wakasa, Yuki Kataoka, et al.Cancer Science|November 24, 2017
DNA replication stress and cancer chemotherapyHiroyuki Kitao, Makoto Iimori, Yuki Kataoka, et al.Scientific Reports|May 30, 2019
Cytotoxicity of trifluridine correlates with the thymidine kinase 1 expression levelYuki Kataoka, Makoto Iimori, Shinichiro Niimi, et al.Cancer Chemotherapy and Pharmacology|April 24, 2020
Detection of trifluridine in tumors of patients with metastatic colorectal cancer treated with trifluridine/tipiracilYoshiaki Fujimoto, Ryota Nakanishi, Mamoru Nukatsuka, et al.Plos One|November 3, 2011
Tissue-restricted expression of Nrf2 and its target genes in zebrafish with gene-specific variations in the induction profilesHitomi Nakajima, Yaeko Nakajima-Takagi, Tadayuki Tsujita, et al.Cancer Science|November 3, 2020
dUTPase inhibition confers susceptibility to a thymidylate synthase inhibitor in DNA-repair-defective human cancer cellsTatsushi Yokogawa, Wakako Yano, Sayaka Tsukioka, et al.Molecular Cancer Research : MCR|June 6, 2018
Thymidine Kinase 1 Loss Confers Trifluridine Resistance without Affecting 5-Fluorouracil Metabolism and CytotoxicityKeitaro Edahiro, Makoto Iimori, Takashi Kobunai, et al.Molecular Cancer Research : MCR|May 30, 2020
DNA Replication Stress Induced by Trifluridine Determines Tumor Cell Fate According to p53 StatusYuki Kataoka, Makoto Iimori, Ryo Fujisawa, et al.Molecular Cancer Therapeutics|May 12, 2018
TAS-114, a First-in-Class Dual dUTPase/DPD Inhibitor, Demonstrates Potential to Improve Therapeutic Efficacy of Fluoropyrimidine-Based ChemotherapyWakako Yano, Tatsushi Yokogawa, Takeshi Wakasa, et al.Pageof 2