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American Journal of Physiology. Endocrinology and Metabolism|October 11, 2012
MicroRNA-494 regulates mitochondrial biogenesis in skeletal muscle through mitochondrial transcription factor A and Forkhead box j3Hirotaka Yamamoto, Katsutaro Morino, Yoshihiko Nishio, et al.
Molecular and Cellular Biology|May 23, 2007
Myosin 5a is an insulin-stimulated Akt2 (protein kinase Bbeta) substrate modulating GLUT4 vesicle translocationTakeshi Yoshizaki, Takeshi Imamura, Jennie L Babendure, et al.
Proceedings of the National Academy of Sciences of the United States of America|May 1, 2008
Beta-Arrestin-1 mediates glucagon-like peptide-1 signaling to insulin secretion in cultured pancreatic beta cellsNoriyuki Sonoda, Takeshi Imamura, Takeshi Yoshizaki, et al.
The Journal of Biological Chemistry|August 1, 2007
Tumor necrosis factor receptor-1 can function through a G alpha q/11-beta-arrestin-1 signaling complexYuji Kawamata, Takeshi Imamura, Jennie L Babendure, et al.
International Journal of Molecular Medicine|March 23, 2011
Postprandial activation of protein kinase Cµ regulates the expression of adipocytokines via the transcription factor AP-2βMotoyuki Kondo, Satoshi Ugi, Katsutaro Morino, et al.
American Journal of Physiology. Endocrinology and Metabolism|March 29, 2007
Suppression of PPAR-gamma attenuates insulin-stimulated glucose uptake by affecting both GLUT1 and GLUT4 in 3T3-L1 adipocytesWei Liao, M T Audrey Nguyen, Takeshi Yoshizaki, et al.
The Journal of Biological Chemistry|March 16, 2004
Protein phosphatase-2C alpha as a positive regulator of insulin sensitivity through direct activation of phosphatidylinositol 3-kinase in 3T3-L1 adipocytesTakeshi Yoshizaki, Hiroshi Maegawa, Katsuya Egawa, et al.
Molecular and Cellular Biology|September 16, 2004
Protein phosphatase 2A negatively regulates insulin's metabolic signaling pathway by inhibiting Akt (protein kinase B) activity in 3T3-L1 adipocytesSatoshi Ugi, Takeshi Imamura, Hiroshi Maegawa, et al.
The Journal of Biological Chemistry|July 31, 2002
Membrane localization of 3-phosphoinositide-dependent protein kinase-1 stimulates activities of Akt and atypical protein kinase C but does not stimulate glucose transport and glycogen synthesis in 3T3-L1 adipocytesKatsuya Egawa, Hiroshi Maegawa, Kun Shi, et al.
Endocrine Journal|June 10, 2009
The onset of diabetes in three out of four sisters: a Japanese family with type 1 diabetes. A case reportAkio Kishi, Yumiko Kawabata, Satoshi Ugi, et al.
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