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Neuroscience Research|February 27, 2018
Loss of GPRC5B impairs synapse formation of Purkinje cells with cerebellar nuclear neurons and disrupts cerebellar synaptic plasticity and motor learningTakamitsu Sano, Ayako Kohyama-Koganeya, Masami O Kinoshita, et al.Plos One|November 18, 2016
Mammalian-Specific Central Myelin Protein Opalin Is Redundant for Normal Myelination: Structural and Behavioral AssessmentsFumio Yoshikawa, Yumi Sato, Koujiro Tohyama, et al.The Journal of Biological Chemistry|December 27, 2005
Formation of tau inclusions in knock-in mice with familial Alzheimer disease (FAD) mutation of presenilin 1 (PS1)Kentaro Tanemura, Du-Hua Chui, Tetsuya Fukuda, et al.Proceedings of the National Academy of Sciences of the United States of America|October 9, 2002
Tau filament formation and associative memory deficit in aged mice expressing mutant (R406W) human tauYoshitaka Tatebayashi, Tomohiro Miyasaka, De-Hua Chui, et al.Iscience|July 5, 2022
Slitrk2 deficiency causes hyperactivity with altered vestibular function and serotonergic dysregulationKei-Ichi Katayama, Naoko Morimura, Katsunori Kobayashi, et al.Human Molecular Genetics|August 8, 2006
Mitochondrial dysfunction and tau hyperphosphorylation in Ts1Cje, a mouse model for Down syndromeEbrahim Abdul Shukkur, Atsushi Shimohata, Takumi Akagi, et al.Human Molecular Genetics|May 23, 2002
Targeted disruption of the Epm2a gene causes formation of Lafora inclusion bodies, neurodegeneration, ataxia, myoclonus epilepsy and impaired behavioral response in miceSubramaniam Ganesh, Antonio V Delgado-Escueta, Toshiro Sakamoto, et al.Pageof 4