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Journal of Bone and Mineral Research : the Official Journal of the American Society for Bone and Mineral Research|February 9, 2017
Connexin43 Mutant Patient-Derived Induced Pluripotent Stem Cells Exhibit Altered Differentiation PotentialJessica L Esseltine, Qing Shao, Courtney Brooks, et al.Disease Models & Mechanisms|March 5, 2009
Oogenesis defects in a mutant mouse model of oculodentodigital dysplasiaDan Tong, Deanne Colley, Renee Thoo, et al.Biology of Reproduction|January 30, 2009
A dominant loss-of-function GJA1 (Cx43) mutant impairs parturition in the mouseDan Tong, Xuerong Lu, Hong-Xing Wang, et al.Elife|October 14, 2020
Single-cell dynamics of pannexin-1-facilitated programmed ATP loss during apoptosisHiromi Imamura, Shuichiro Sakamoto, Tomoki Yoshida, et al.Bone|April 23, 2020
Effects of reduced connexin43 function on skull development in the Cx43I130T/+ mutant mouse that models oculodentodigital dysplasiaSommer E Jarvis, Jae Eun Lee, Elizabeth Jewlal, et al.Journal of Bone and Mineral Research : the Official Journal of the American Society for Bone and Mineral Research|September 15, 2011
Pannexin 3 is a novel target for Runx2, expressed by osteoblasts and mature growth plate chondrocytesStephen R Bond, Alice Lau, Silvia Penuela, et al.Community Mental Health Journal|May 14, 2020
Social Prescribing for Individuals Living with Mental Illness in an Australian Community Setting: A Pilot StudyChristina Aggar, Tamsin Thomas, Christopher Gordon, et al.International Journal of Environmental Research and Public Health|February 9, 2021
Stepped-Wedge Cluster Randomised Trial of Social Prescribing of Forest Therapy for Quality of Life and Biopsychosocial Wellbeing in Community-Living Australian Adults with Mental Illness: ProtocolTamsin Thomas, James Baker, Debbie Massey, et al.Molecular Biology of the Cell|May 27, 2016
Specific functional pathologies of Cx43 mutations associated with oculodentodigital dysplasiaJohn J Kelly, Jessica L Esseltine, Qing Shao, et al.The Journal of Biological Chemistry|June 4, 2002
Retroviral delivery of connexin genes to human breast tumor cells inhibits in vivo tumor growth by a mechanism that is independent of significant gap junctional intercellular communicationHong Qin, Qing Shao, Heather Curtis, et al.Pageof 15